The Pseudomonas quinolne signal regulates rhl quorum sensing in Pseudomonas aeruginosa
The opportunistic pathogen Pseudomonas aeruginosa uses intercellular signals to control the density-dependent expression of many virulence factors. The las and rhl quorum-sensing systems function, respectively, through the autoinducers N-(3-oxododecanoyl)-L-homoserine lactone and N-butyryl-L-homoser...
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| Published in | Journal of bacteriology Vol. 182; no. 10; p. 2702 |
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| Main Authors | , , |
| Format | Journal Article |
| Language | English |
| Published |
Washington
American Society for Microbiology
01.05.2000
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| Subjects | |
| Online Access | Get full text |
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| Abstract | The opportunistic pathogen Pseudomonas aeruginosa uses intercellular signals to control the density-dependent expression of many virulence factors. The las and rhl quorum-sensing systems function, respectively, through the autoinducers N-(3-oxododecanoyl)-L-homoserine lactone and N-butyryl-L-homoserine lactone (C4-HSL), which are known to positively regulate the transcription of the elastase-encoding gene, lasB. |
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| AbstractList | The opportunistic pathogen Pseudomonas aeruginosa uses intercellular signals to control the density-dependent expression of many virulence factors. The las and rhl quorum-sensing systems function, respectively, through the autoinducers N-(3-oxododecanoyl)-L-homoserine lactone and N-butyryl-L-homoserine lactone (C4-HSL), which are known to positively regulate the transcription of the elastase-encoding gene, lasB. |
| Author | McKnight, Susan L Iglewski, Barbara H Pesci, Everett C |
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| Title | The Pseudomonas quinolne signal regulates rhl quorum sensing in Pseudomonas aeruginosa |
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