Regulation of Mitochondria Oxidative Metabolism by Tumor Suppressor FLCN
Birt-Hogg-Dube (BHD) syndrome is a hereditary hamartoma syndrome that predisposes patients to develop hair follicle tumors, lung cysts, and kidney cancer. Genetic studies of BHD patients have uncovered the causative gene, FLCN, but its function is incompletely understood. Here, Hasumi et al conduct...
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| Published in | JNCI : Journal of the National Cancer Institute Vol. 104; no. 22; p. 1750 |
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| Main Authors | , , , , , , , , , , |
| Format | Journal Article |
| Language | English |
| Published |
Oxford
Oxford Publishing Limited (England)
21.11.2012
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| Subjects | |
| Online Access | Get full text |
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| Abstract | Birt-Hogg-Dube (BHD) syndrome is a hereditary hamartoma syndrome that predisposes patients to develop hair follicle tumors, lung cysts, and kidney cancer. Genetic studies of BHD patients have uncovered the causative gene, FLCN, but its function is incompletely understood. Here, Hasumi et al conduct metabolic assays using FLCN-deficient muscle. |
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| AbstractList | Birt-Hogg-Dube (BHD) syndrome is a hereditary hamartoma syndrome that predisposes patients to develop hair follicle tumors, lung cysts, and kidney cancer. Genetic studies of BHD patients have uncovered the causative gene, FLCN, but its function is incompletely understood. Here, Hasumi et al conduct metabolic assays using FLCN-deficient muscle. |
| Author | Baba, Masaya Hasumi, Yukiko Linehan, W Marston Oh, Hyoungbin Hughes, Robert M Klein, Mara E Hasumi, Hisashi Schmidt, Laura S Huang, Ying Takikita, Shoichi Nagashima, Kunio |
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| Title | Regulation of Mitochondria Oxidative Metabolism by Tumor Suppressor FLCN |
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