Finerenone y su papel en la enfermedad renal diabética. Estado del arte
Diabetic kidney disease represents the leading cause of end-stage kidney disease and its presence directly impacts cardiovascular risk and mortality in individuals with diabetes mellitus. Since albuminuria is the main marker of DKD, its reduction has been established in recent years as one of the th...
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Published in | Archivos de medicina Vol. 18; no. 1 |
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Main Authors | , , , , , , , , |
Format | Journal Article |
Language | Spanish |
Published |
2022
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Subjects | |
Online Access | Get full text |
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Summary: | Diabetic kidney disease represents the leading cause of end-stage kidney disease and its presence directly impacts cardiovascular risk and mortality in individuals with diabetes mellitus. Since albuminuria is the main marker of DKD, its reduction has been established in recent years as one of the therapeutic objectives to impact the evolution of the disease. Finerenone, a mineralocorticoid receptor antagonist, has been described in several recent studies as a drug whose mechanism of action could contribute to reducing the progression of DKD, reducing the deterioration of eGFR and albuminuria with an adequate profile of safety in patients with all stages of kidney disease.
La enfermedad renal diabética representa la primera causa de enfermedad renal terminal y su presencia impacta directamente en el riesgo cardiovascular y la mortalidad de los individuos con diabetes mellitus. Siendo la albuminuria el principal marcador de la ERD su reducción se ha constituido en los últimos años como uno de los objetivos terapéuticos para impactar en la evolución de la enfermedad. El finerenone, un antagonista de los receptores mineralocorticoides, se ha descrito en diversos estudios recientes como un fármaco cuyo mecanismo de acción podría contribuir a la reducción de la progresión de la ERD, disminuyendo el deterioro de la TFGe y la albuminuria con un adecuado perfil de seguridad en pacientes en todos los estadios de la enfermedad renal. |
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ISSN: | 1698-9465 1698-9465 |