Impaired M-current in KCNQ2 Encephalopathy Evokes Dyshomeostatic Modulation of Excitability

“The authors have withdrawn their manuscript whilst they perform additional experiments to test some of their conclusions further. Therefore, the authors do not wish this work to be cited as reference for the project. If you have any questions, please contact the corresponding authors.” Competing In...

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Bibliographic Details
Published inbioRxiv
Main Authors Simkin, Dina, Searl, Timothy J, Piyevsky, Brandon N, rest, Marc, Williams, Luis A, Joshi, Vaibhav, Zhang, Hongkang, Ryan, Steven J, Schwake, Michael, Robertson, Gabriella L, Penzes, Peter, Laux, Linda C, Mcmanus, Owen B, Dempsey, Graham T, Millichap, John J, George, Alfred L, Kiskinis, Evangelos
Format Paper
LanguageEnglish
Published Cold Spring Harbor Cold Spring Harbor Laboratory Press 22.09.2020
Subjects
Action potential
Afterhyperpolarization
Age
Calcium channels
Calcium conductance
Encephalopathy
Epilepsy
Excitability
Firing pattern
KCNQ2 protein
Mutation
Neonates
Neurodevelopment
Neurons
Pluripotency
Potassium channels
Potassium channels (calcium-gated)
Potassium channels (voltage-gated)
Potassium conductance
Seizures
Stem cells
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Summary:“The authors have withdrawn their manuscript whilst they perform additional experiments to test some of their conclusions further. Therefore, the authors do not wish this work to be cited as reference for the project. If you have any questions, please contact the corresponding authors.” Competing Interest Statement The authors have declared no competing interest.
DOI:10.1101/538371