Left ventricular wall stress normalization in chronic pressure-overloaded heart : A mathematical model study

It is generally accepted that the left ventricle (LV) hypertrophies (LVH) to normalize systolic wall stress (sigmas) in chronic presure overload. However, LV filling pressure (Pv) may be elecated as well, supporting the alternative hypothesis of end-diastolic wall stress (sigmad) normalization in LV...

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Published inAmerican journal of physiology. Heart and circulatory physiology Vol. 48; no. 3; pp. H1120 - H1127
Main Authors SEGERS, Patrick, STERGIOPULOS, Nikos, SCHREUDER, J. A. N. J, WESTERHOF, Berend E, WESTERHOF, Nico
Format Journal Article
LanguageEnglish
Published Bethesda, MD American Physiological Society 01.09.2000
Subjects
Aging
Arterial hypertension. Arterial hypotension
Biological and medical sciences
Blood and lymphatic vessels
Blood vessels
Cardiology. Vascular system
Clinical manifestations. Epidemiology. Investigative techniques. Etiology
Heart
Hypertension
Mathematical models
Medical sciences
Heart
Hypertension
Fissipedia
Carnivora
Cardiovascular disease
Modeling
Compliance(volume pressure)
Stress
Left ventricle
Elastance
Vertebrata
Compensatory hypertrophy
Mammalia
Overload
Animal
Cat
Circulatory system
Mathematical model
Vascular wall
Dog
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Summary:It is generally accepted that the left ventricle (LV) hypertrophies (LVH) to normalize systolic wall stress (sigmas) in chronic presure overload. However, LV filling pressure (Pv) may be elecated as well, supporting the alternative hypothesis of end-diastolic wall stress (sigmad) normalization in LVH.
ISSN:0363-6135
1522-1539