Effect of insulin-like growth factor I on in vivo intestinal absorption of D-galactose in cirrhotic rats

Insulin-like growth factor I (IGF-I) bioavailability is reduced in liver cirrhosis, a condition frequently associated with malnutrition. We have analyzed in vivo absorption of D-galactose by jejunal loops in rats with CCl4-induced liver cirrhosis and the influence of IGF-I on intesinal sugar transpo...

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Published inAmerican journal of physiology: Gastrointestinal and liver physiology Vol. 39; no. 1; pp. G37 - G42
Main Authors CASTILLA-CORTAZAR, I, PICARDI, A, TOSAR, A, AINZUA, J, URDANETA, E, GARCIA, M, PASCUAL, M, QUIROGA, J, PRIETO, J
Format Journal Article
LanguageEnglish
Published Bethesda, MD American Physiological Society 1999
Subjects
Biological and medical sciences
Gastroenterology. Liver. Pancreas. Abdomen
Glucose
Liver cirrhosis
Liver. Biliary tract. Portal circulation. Exocrine pancreas
Malnutrition
Medical sciences
Other diseases. Semiology
Rodents
Animal model
Rat
Digestive system
Rodentia
Nutrition disorder
Hepatic disease
Insulin like growth factor 1
Small intestine
Microvilli
Prevention
Cirrhosis
Vertebrata
Mammalia
Absorption
Jejunum
Animal
Digestive diseases
Carbohydrate
Galactose
Malnutrition
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Summary:Insulin-like growth factor I (IGF-I) bioavailability is reduced in liver cirrhosis, a condition frequently associated with malnutrition. We have analyzed in vivo absorption of D-galactose by jejunal loops in rats with CCl4-induced liver cirrhosis and the influence of IGF-I on intesinal sugar transport in this disease.
ISSN:0193-1857
1522-1547