The cytoprotective effect of prostaglandin E2 in duodenal ulcers

Intragastric administration of a synthetic analog of prostaglandin E2 to patients with duodenal ulcers results in depression of the gastric acid production and in reduction of potassium level, leads to an elevation of the sialic acid debit and a rise of sodium concentration, not essentially changing...

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Bibliographic Details
Published inLaboratornoe delo no. 7; p. 45
Main Authors Skliarov, E Ia, Panasiuk, E N, Skliarov, A Ia
Format Journal Article
LanguageRussian
Published Russia (Federation) 1989
Subjects
Adolescent
Adult
Aged
Dinoprostone - therapeutic use
Duodenal Ulcer - drug therapy
Duodenal Ulcer - physiopathology
Electrolytes - analysis
Female
Gastric Acid - secretion
Humans
Male
Middle Aged
Pepsin A - analysis
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Summary:Intragastric administration of a synthetic analog of prostaglandin E2 to patients with duodenal ulcers results in depression of the gastric acid production and in reduction of potassium level, leads to an elevation of the sialic acid debit and a rise of sodium concentration, not essentially changing calcium, chlorides, and pepsin levels. A combined analysis of gastric acid production and measurements of sialic acids, sodium and potassium detect the imbalance of the mucoid and bicarbonate secretion in peptic ulcers, this helping predict the disease course.
ISSN:0023-6748