Neurological manifestations of anti-phospholipid (Hughes) syndrome

In the 21 years since the description of the antiphospholipid syndrome (APS), neurological features have become recognised as some of the most important and common aspects of the syndrome. Recognition of the relationship between APS and central nervous system impairment could open the door to improv...

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Published inClinical and experimental rheumatology Vol. 22; no. 6; pp. 771 - 775
Main Authors ANTUNES, M. C, D'CRUZ, D, HUGHES, G. R. V
Format Journal Article
LanguageEnglish
Published Pisa Clinical and Experimental Rheumatology 01.11.2004
Subjects
Antiphospholipid Syndrome - complications
Antiphospholipid Syndrome - immunology
Antiphospholipid Syndrome - physiopathology
Biological and medical sciences
Hematologic and hematopoietic diseases
Humans
Medical sciences
Nervous System Diseases - etiology
Nervous System Diseases - immunology
Nervous System Diseases - physiopathology
Platelet diseases and coagulopathies
Thrombosis - etiology
Thrombosis - immunology
Thrombosis - physiopathology
Autoimmunity
Hypercoagulability
anticardiolipin antibodies
Circulating anticoagulant
Antiphospholipid antibody syndrome
Antibody
Cardiovascular disease
Autoimmune disease
Hemopathy
lupus anticoagulant
Thrombosis
Vascular disease
Platelet
Coagulopathy
Antiphospholipid syndrome
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Summary:In the 21 years since the description of the antiphospholipid syndrome (APS), neurological features have become recognised as some of the most important and common aspects of the syndrome. Recognition of the relationship between APS and central nervous system impairment could open the door to improved treatment and outcomes in some neurological syndromes.
Bibliography:ObjectType-Article-2
SourceType-Scholarly Journals-1
ObjectType-Feature-3
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ObjectType-Review-1
ISSN:0392-856X
1593-098X