Induction and maintenance of increased VEGF protein by chronic motor nerve stimulation in skeletal muscle

Vascular endothelial growth factor (VEGF) causes endothelial cell proliferation in vitro and angiogenesis in vivo. Glycolytic skeletal muscles have a lower capillary density than oxidative muscles but can increase their capillary density and convert to a more oxidative phenotype when subject to chro...

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Published inAmerican journal of physiology. Heart and circulatory physiology Vol. 43; no. 3; pp. H860 - H867
Main Authors ANNEX, B. H, TORGAN, C. E, PENGNIAN LIN, TAYLOR, D. A, THOMPSON, M. A, PETERS, K. G, KRAUS, W. E
Format Journal Article
LanguageEnglish
Published Bethesda, MD American Physiological Society 01.03.1998
Subjects
Biological and medical sciences
Cellular biology
Circulatory system
Fundamental and applied biological sciences. Psychology
Muscular system
Nervous system
Proteins
Rabbits
Striated muscle. Tendons
Vertebrates: osteoarticular system, musculoskeletal system
Angiogenesis
Vertebrata
Mammalia
Vascular endothelium growth factor
Oxidative phosphorylation
Blood vessel
Glycolysis
Rabbit
Circulatory system
Lagomorpha
Striated muscle
Vascularization
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Summary:Vascular endothelial growth factor (VEGF) causes endothelial cell proliferation in vitro and angiogenesis in vivo. Glycolytic skeletal muscles have a lower capillary density than oxidative muscles but can increase their capillary density and convert to a more oxidative phenotype when subject to chronic motor nerve stimulation (CMNS).
ISSN:0363-6135
1522-1539