BUB1-bling over with Possibilities

Abstract In a new report, Nyati et al. identified a previously undetected participant mediating both canonical signaling, i.e., TGF-β receptor kinase mediated, and non-canonical signaling, budding uninhibited by benzimidazole 1 (BUB1).

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Bibliographic Details
Published inNeoplasia (New York, N.Y.) Vol. 17; no. 2; pp. 153 - 154
Main Author Barcellos-Hoff, Mary Helen
Format Journal Article
LanguageEnglish
Published United States Elsevier Inc 01.02.2015
Neoplasia Press
Elsevier
Subjects
Animals
Humans
Oncology
Protein-Serine-Threonine Kinases - metabolism
Receptors, Transforming Growth Factor beta - metabolism
Signal Transduction - physiology
Smad Proteins, Receptor-Regulated - metabolism
Transforming Growth Factor beta - physiology
TGFβ receptor type I
TGFBRI
transforming growth factor–β
TGFβ
BUB1
budding uninhibited by benzimidazole 1
TGFBRII
TGFβ receptor type II
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Summary:Abstract In a new report, Nyati et al. identified a previously undetected participant mediating both canonical signaling, i.e., TGF-β receptor kinase mediated, and non-canonical signaling, budding uninhibited by benzimidazole 1 (BUB1).
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ISSN:1476-5586
1522-8002
1476-5586
1522-8002
DOI:10.1016/j.neo.2015.01.002