Global premature mortality due to anthropogenic outdoor air pollution and the contribution of past climate change

• Published in: Environmental research letters Vol. 8; no. 3; pp. 1 - 11
• Main Authors: Silva, Raquel A, West, J Jason, Zhang, Yuqiang, Anenberg, Susan C, Lamarque, Jean-François, Shindell, Drew T, Collins, William J, Dalsoren, Stig, Faluvegi, Greg, Folberth, Gerd, Horowitz, Larry W, Nagashima, Tatsuya, Naik, Vaishali, Rumbold, Steven, Skeie, Ragnhild, Sudo, Kengo, Takemura, Toshihiko, Bergmann, Daniel, Cameron-Smith, Philip, Cionni, Irene, Doherty, Ruth M, Eyring, Veronika, Josse, Beatrice, MacKenzie, I A, Plummer, David, Righi, Mattia, Stevenson, David S, Strode, Sarah, Szopa, Sophie, Zeng, Guang
• Format: Journal Article
• Language: English
• Published: Goddard Space Flight Center IOP Publishing 01.09.2013
• Subjects: Air pollution; Air quality; Anthropogenic factors; Atmospheric chemistry; Bioclimatology; Climate change; Climate effects; Climate models; Confidence intervals; Death; Ecology, environment; Environment Pollution; Estimates; Fatalities; Health; human health; Life Sciences; Lung cancer; Lung diseases; Meteorology And Climatology; Mortality; Ozone; Particulate emissions; Particulate matter; Pollution control; Premature mortality; Response functions; Santé publique et épidémiologie; Uncertainty; Particulate Matter; Ozone; Premature Mortality; Air Pollution; Climate Change; Human Health; premature mortality; particulate matter; air pollution; ozone; human health; climate change
• ISSN: 1748-9326; 1748-9326
• DOI: 10.1088/1748-9326/8/3/034005

Increased concentrations of ozone and fine particulate matter (PM2.5) since preindustrial times reflect increased emissions, but also contributions of past climate change. Here we use modeled concentrations from an ensemble of chemistry-climate models to estimate the global burden of anthropogenic outdoor air pollution on present-day premature human mortality, and the component of that burden attributable to past climate change. Using simulated concentrations for 2000 and 1850 and concentration-response functions (CRFs), we estimate that, at present, 470 000 (95% confidence interval, 140 000 to 900 000) premature respiratory deaths are associated globally and annually with anthropogenic ozone, and 2.1 (1.3 to 3.0) million deaths with anthropogenic PM2.5-related cardiopulmonary diseases (93%) and lung cancer (7%). These estimates are smaller than ones from previous studies because we use modeled 1850 air pollution rather than a counterfactual low concentration, and because of different emissions. Uncertainty in CRFs contributes more to overall uncertainty than the spread of model results. Mortality attributed to the effects of past climate change on air quality is considerably smaller than the global burden: 1500 (−20 000 to 27 000) deaths yr−1 due to ozone and 2200 (−350 000 to 140 000) due to PM2.5. The small multi-model means are coincidental, as there are larger ranges of results for individual models, reflected in the large uncertainties, with some models suggesting that past climate change has reduced air pollution mortality.