The Golgi apparatus: Site for convergence of COVID-19 brain fog and Alzheimer's disease?

Keywords: Alzheimer's disease, Autophagy, Brain fog, Golgi, Glycolipids, Glycosylation, GRASP55, GRASP65, Heparan sulfate proteoglycans, Intracellular trafficking, Myelin, O-GlcNAcylation, Phosphorylation, SARS-CoV-2 infection, Secretion

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Bibliographic Details
Published inMolecular neurodegeneration Vol. 17; no. 1; p. 67
Main Authors Wang, Yanzhuang, Gandy, Sam
Format Journal Article
LanguageEnglish
Published England BioMed Central Ltd 21.10.2022
BioMed Central
BMC
Subjects
Advertising executives
Alzheimer Disease - metabolism
Alzheimer's disease
Autophagy
Brain
Brain fog
Coronaviruses
COVID-19
Endoplasmic reticulum
Fibrils
Glycolipids
Glycosylation
Golgi
Golgi Apparatus
Heparan sulfate
Humans
Infection
Infections
Kinases
Lysosomes
Membrane lipids
Membrane vesicles
Neurodegenerative diseases
Neuropathology
Neurotoxicity
Phagosomes
Phosphorylation
Protein Transport
Proteins
Severe acute respiratory syndrome coronavirus 2
Structural proteins
Phosphorylation
Alzheimer’s disease
Myelin
Secretion
Brain fog
SARS-CoV-2 infection
Glycosylation
Autophagy
GRASP65
GRASP55
Glycolipids
Intracellular trafficking
Heparan sulfate proteoglycans
O-GlcNAcylation
Golgi
Online AccessGet full text

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More Information
Summary:Keywords: Alzheimer's disease, Autophagy, Brain fog, Golgi, Glycolipids, Glycosylation, GRASP55, GRASP65, Heparan sulfate proteoglycans, Intracellular trafficking, Myelin, O-GlcNAcylation, Phosphorylation, SARS-CoV-2 infection, Secretion
Bibliography:ObjectType-Article-1
SourceType-Scholarly Journals-1
ObjectType-Feature-2
content type line 23
ISSN:1750-1326
1750-1326
DOI:10.1186/s13024-022-00568-2