Basal ganglia calcification as a putative cause for cognitive decline

Basal ganglia calcifications (BGC) may be present in various medical conditions, such as infections, metabolic, psychiatric and neurological diseases, associated with different etiologies and clinical outcomes, including parkinsonism, psychosis, mood swings and dementia. A literature review was perf...

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Published inDementia & neuropsychologia Vol. 7; no. 2; pp. 151 - 154
Main Authors de Oliveira, João Ricardo Mendes, de Oliveira, Matheus Fernandes
Format Journal Article
LanguageEnglish
Published Brazil Associação de Neurologia Cognitiva e do Comportamento 01.06.2013
Associação Neurologia Cognitiva e do Comportamento
Subjects
basal ganglia
calcinosis
CLINICAL NEUROLOGY
dementia
GERIATRICS & GERONTOLOGY
HEALTH CARE SCIENCES & SERVICES
MEDICINE, GENERAL & INTERNAL
NURSING
PSYCHIATRY
PSYCHOLOGY
Views & Reviews
dementia
basal ganglia
calcinosis
núcleos da base
calcificações
demência
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Summary:Basal ganglia calcifications (BGC) may be present in various medical conditions, such as infections, metabolic, psychiatric and neurological diseases, associated with different etiologies and clinical outcomes, including parkinsonism, psychosis, mood swings and dementia. A literature review was performed highlighting the main neuropsychological findings of BGC, with particular attention to clinical reports of cognitive decline. Neuroimaging studies combined with neuropsychological analysis show that some patients have shown progressive disturbances of selective attention, declarative memory and verbal perseveration. Therefore, the calcification process might represent a putative cause for dementia syndromes, suggesting a probable link among calcinosis, the aging process and eventually with neuronal death. The increasing number of reports available will foster a necessary discussion about cerebral calcinosis and its role in determining symptomatology in dementia patients.
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ISSN:1980-5764
1980-5764
DOI:10.1590/s1980-57642013dn70200003