Neuroinflammation after traumatic brain injury: Opportunities for therapeutic intervention

Highlight ► Neuroinflammation is a key secondary injury mechanism following traumatic brain injury, and selective inhibition of these pathways result in significant neuroprotection.

Saved in:
Bibliographic Details
Published inBrain, behavior, and immunity Vol. 26; no. 8; pp. 1191 - 1201
Main Authors Kumar, Alok, Loane, David J
Format Journal Article
LanguageEnglish
Published Netherlands Elsevier Inc 01.11.2012
Subjects
Allergy and Immunology
Animals
Anti-Inflammatory Agents - therapeutic use
Brain Injuries - complications
Brain Injuries - drug therapy
Brain Injuries - physiopathology
Brain injury
Chronic neurodegeneration
Clinical trials
Disease Models, Animal
Encephalitis - drug therapy
Encephalitis - etiology
Humans
Immunosuppressive agents
Inflammation
Microglia
Morbidity
Mortality
Neuroinflammation
Neurological diseases
Neuroprotection
Neuroprotective Agents - therapeutic use
Neurotoxicity
Psychiatry
Regeneration
Therapeutic applications
Traumatic brain injury
Neuroprotection
Neuroinflammation
Traumatic brain injury
Chronic neurodegeneration
Microglia
Online AccessGet full text

Cover

More Information
Summary:Highlight ► Neuroinflammation is a key secondary injury mechanism following traumatic brain injury, and selective inhibition of these pathways result in significant neuroprotection.
Bibliography:ObjectType-Article-2
SourceType-Scholarly Journals-1
ObjectType-Feature-1
content type line 23
ISSN:0889-1591
1090-2139
DOI:10.1016/j.bbi.2012.06.008