Is Recurrent Perseveration a Product of Deafferented Functional Systems with Otherwise Normal Post-Activation Decay Rates?

Recent work in neuropsychology, clinical aphasiology and neuropharmacology have presented evidence that the causative substrates of recurrent perseveration in adults with aphasia are more recondite and subject to distinct interpretations than originally thought. This article will discuss and evaluat...

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Bibliographic Details
Published inClinical linguistics & phonetics Vol. 25; no. 11-12; pp. 1066 - 1073
Main Authors Buckingham, Hugh W, Buckingham, Sarah S
Format Journal Article
LanguageEnglish
Published England Informa Healthcare 01.11.2011
Subjects
Adults
Afferent Pathways - physiopathology
Aphasia
Aphasia - drug therapy
Aphasia - etiology
Aphasia - physiopathology
Behavior Therapy
Brain Damage, Chronic - complications
Brain Damage, Chronic - drug therapy
Brain Damage, Chronic - physiopathology
Cholinergic Agents - therapeutic use
Dopamine Agents - therapeutic use
Drug Therapy
Etiology
Evidence
Executive Function
Humans
Linguistics
Models, Neurological
Neurological Organization
Neuropsychological Tests
Neuropsychology
Pharmacology
Psychomotor Performance
Recurrence
Repetition
Stereotyped Behavior - physiology
Theory Practice Relationship
Verbal Behavior - physiology
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Summary:Recent work in neuropsychology, clinical aphasiology and neuropharmacology have presented evidence that the causative substrates of recurrent perseveration in adults with aphasia are more recondite and subject to distinct interpretations than originally thought. This article will discuss and evaluate how various proposals from theory, from the clinic and from drug therapy interact and compete in the search for a cause or causes of recurrent perseveration.
ISSN:0269-9206
1464-5076
DOI:10.3109/02699206.2011.616982