Trismus due to myotonia associated with hyperadrenocorticism in a dog

We present the report of trismus due to hyperadrenocorticism-associated myotonia diagnosed by electromyography in a dog. An intact female Miniature Dachshund, 13 years and 9 months old, presented with stiff gait and trismus as well as polyuria and polydipsia. Abdominal ultrasonography showed enlarge...

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Published inJournal of Veterinary Medical Science Vol. 85; no. 8; pp. 876 - 879
Main Authors YOKOTA, Shunya, KOBATAKE, Yui, MAEKAWA, Masahiro, TAKASHIMA, Satoshi, NISHII, Naohito
Format Journal Article
LanguageEnglish
Published Japan JAPANESE SOCIETY OF VETERINARY SCIENCE 2023
Japan Science and Technology Agency
The Japanese Society of Veterinary Science
Subjects
Adrenal glands
Adrenocorticotropic hormone
dog
Electromyography
Gait
hyperadrenocorticism
Internal Medicine
Myotonia
Polydipsia
Polyuria
Temporalis muscle
trismus
myotonia
trismus
hyperadrenocorticism
dog
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Summary:We present the report of trismus due to hyperadrenocorticism-associated myotonia diagnosed by electromyography in a dog. An intact female Miniature Dachshund, 13 years and 9 months old, presented with stiff gait and trismus as well as polyuria and polydipsia. Abdominal ultrasonography showed enlarged adrenal glands. An adrenocorticotropic hormone stimulation test revealed an exaggerated response. Based on these findings, this case was diagnosed with hyperadrenocorticism. Electromyography revealed myotonic discharge in the temporalis muscle and limbs. Therefore, trismus was considered to be caused by hyperadrenocorticism-associated myotonia, and the case was treated with oral trilostane (1.3 mg/kg, once daily). During the 4-month follow-up period, despite the partial improvement in stiff gait, trismus did not recover. Long-term data on more cases are warranted to assess the prognosis and clinical characteristics of trismus due to hyperadrenocorticism-associated myotonia.
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content type line 23
ISSN:0916-7250
1347-7439
DOI:10.1292/jvms.23-0103