The 1918 Influenza Pandemic and Subsequent Health Outcomes: An Analysis of SIPP Data
The fetal-origins hypothesis has recently achieved "textbook" status in medicine (e.g. Rudolph, 2003 and Warrell, 2003) and is drawing increased attention for social sciences (e.g., Case, Fertig and Paxton, 2005; Currie and Moretti, 2005). The hypotheses holds that disruptions to fetal nut...
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Published in | The American economic review Vol. 95; no. 2; pp. 258 - 262 |
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Main Authors | , |
Format | Journal Article |
Language | English |
Published |
United States
American Economic Association
01.05.2005
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Subjects | |
Online Access | Get full text |
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Summary: | The fetal-origins hypothesis has recently achieved "textbook" status in medicine (e.g. Rudolph, 2003 and Warrell, 2003) and is drawing increased attention for social sciences (e.g., Case, Fertig and Paxton, 2005; Currie and Moretti, 2005). The hypotheses holds that disruptions to fetal nutrition can exert persistent effects on subsequent health. Recent research has used the 1918 Influenza Pandemic as a natural experiment for studying the effects of fetal health (Almond, 2005). In contrast to typical influenza strains, the 1918 virus disproportionately affects young adults, approximately one-third of pregnant women contracted the debilitating virus. Using data from the Survey of Income and Program Participation (SIPP), this study finds that cohorts in utero during the Pandemic exhibit impaired health outcomes relative to cohorts born a few months earlier or later. That these patterns are manifest 65-80 years after the Pandemic suggests that changes to fetal health can have life-long effects. |
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Bibliography: | ObjectType-Article-1 SourceType-Scholarly Journals-1 ObjectType-Feature-2 content type line 23 ObjectType-Article-2 ObjectType-Feature-1 |
ISSN: | 0002-8282 1944-7981 |
DOI: | 10.1257/000282805774669943 |