Ultrasound Biomicroscopic Analysis of Transient Shallow Anterior Chamber in Vogt-Koyanagi-Harada Syndrome

• Published in: American journal of ophthalmology Vol. 121; no. 6; pp. 720 - 723
• Main Authors: Kawano, Yoh-Ichi, Tawara, Akihiko, Nishioka, Yuko, Suyama, Yayoi, Sakamoto, Hidehisa, Inomata, Hajime
• Format: Journal Article
• Language: English
• Published: New York, NY Elsevier Inc 01.06.1996; Elsevier; Elsevier Limited
• Subjects: Anterior Chamber - diagnostic imaging; Anterior Chamber - pathology; Anterior Eye Segment - diagnostic imaging; Anterior Eye Segment - pathology; Anti-Inflammatory Agents - therapeutic use; Biological and medical sciences; Choroid Diseases - diagnostic imaging; Choroid Diseases - etiology; Ciliary Body - diagnostic imaging; Ciliary Body - pathology; Dexamethasone - therapeutic use; Female; Humans; Medical sciences; Microscopy; Middle Aged; Ophthalmology; Ultrasonography; Uvea diseases; Uveal Diseases - diagnostic imaging; Uveal Diseases - etiology; Uveomeningoencephalitic Syndrome - complications; Uveomeningoencephalitic Syndrome - diagnostic imaging; Uveomeningoencephalitic Syndrome - drug therapy; Sonography; Biomicroscopy; Human; Skin disease; Nervous system diseases; Pigmentation disorder; Pathogenesis; Uvea disease; Transitory; Case study; Eye disease; Athalamia; Vogt Koyanagi uveitis; Central nervous system disease; Meningitis; Anterior chamber; Ultrasound
• ISSN: 0002-9394; 1879-1891
• DOI: 10.1016/S0002-9394(14)70645-2

To evaluate the mechanism of formation of the transient shallow anterior chamber in Vogt-Koyanagi-Harada syndrome. Two patients with Vogt-Koyanagi-Harada syndrome with shallow anterior chambers were examined with an ultrasound biomicroscope. A ciliochoroidal detachment, which was not obvious on ophthalmoscopic examination, was clearly demonstrated in both patients by ultrasound biomicroscopy. The detachment disappeared after systemic corticosteroid therapy. The shallowing of the anterior chamber in two patients with Vogt-Koyanagi-Harada syndrome was caused by suprachoroidal effusion secondary to inflammation of the uvea.