APOL1 , α-thalassemia, and BCL11A variants as a genetic risk profile for progression of chronic kidney disease in sickle cell anemia

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Bibliographic Details
Published inHaematologica (Roma) Vol. 102; no. 1; pp. e1 - e6
Main Authors Saraf, Santosh L., Shah, Binal N., Zhang, Xu, Han, Jin, Tayo, Bamidele O., Abbasi, Taimur, Ostrower, Adam, Guzman, Elizabeth, Molokie, Robert E., Gowhari, Michel, Hassan, Johara, Jain, Shivi, Cooper, Richard S., Machado, Roberto F., Lash, James P., Gordeuk, Victor R.
Format Journal Article
LanguageEnglish
Published Italy Ferrata Storti Foundation 01.01.2017
Subjects
Adult
alpha-Thalassemia - complications
alpha-Thalassemia - genetics
Anemia, Sickle Cell
Apolipoprotein L1 - genetics
Carrier Proteins - genetics
Female
Humans
Male
Nuclear Proteins - genetics
Online Only
Renal Insufficiency, Chronic - etiology
Renal Insufficiency, Chronic - genetics
Risk Factors
Online AccessGet full text

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Bibliography:SourceType-Other Sources-1
ObjectType-Article-1
content type line 63
ObjectType-Correspondence-2
ISSN:0390-6078
1592-8721
DOI:10.3324/haematol.2016.154153