Budd-Chiari–Induced Protein-Losing Enteropathy After Liver Transplantation

Abstract Budd-Chiari syndrome (BCS) is one of the uncommon complications of hepatic venous reconstruction in liver transplantation. Protein-losing enteropathy (PLE) secondary to this event has rarely been described. A 14-year-old girl suffering from acute hepatic failure underwent an emergency livin...

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Bibliographic Details
Published inTransplantation proceedings Vol. 39; no. 5; pp. 1554 - 1557
Main Authors Wong, J, Sin, N.C, Sung, R, Lee, P.S.F, Lee, K.F, Lai, P.B.S
Format Journal Article
LanguageEnglish
Published New York, NY Elsevier Inc 01.06.2007
Elsevier Science
Subjects
Adolescent
Biological and medical sciences
Budd-Chiari Syndrome - diagnosis
Budd-Chiari Syndrome - surgery
Female
Fundamental and applied biological sciences. Psychology
Fundamental immunology
Gastroenterology. Liver. Pancreas. Abdomen
Hepatic Veins - surgery
Humans
Liver Cirrhosis - pathology
Liver Cirrhosis - surgery
Liver Transplantation - adverse effects
Living Donors
Male
Medical sciences
Necrosis
Other diseases. Semiology
Protein-Losing Enteropathies - etiology
Stomach. Duodenum. Small intestine. Colon. Rectum. Anus
Surgery
Surgery (general aspects). Transplantations, organ and tissue grafts. Graft diseases
Tissue, organ and graft immunology
Treatment Outcome
Medicine
Treatment
Digestive system
Intestinal malabsorption
Surgery
Liver
Digestive diseases
Intestinal disease
Graft
Transplantation
Homotransplantation
Protein losing enteropathy
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Summary:Abstract Budd-Chiari syndrome (BCS) is one of the uncommon complications of hepatic venous reconstruction in liver transplantation. Protein-losing enteropathy (PLE) secondary to this event has rarely been described. A 14-year-old girl suffering from acute hepatic failure underwent an emergency living-related liver transplantation and developed BCS 1 year later. Her condition has been managed with several sessions of hepatic venoplasty. On one occasion, she suffered septicemia and severe diarrhea, passing large amount of fibrinoid material. The diagnosis of PLE was made clinically, which resolved immediately after reestablishment of hepatic venous patency by balloon venoplasty. This observation suggested that BCS was responsible for PLE in this patient. Prompt diagnosis and early intervention for this life-threatening condition is essential.
Bibliography:ObjectType-Case Study-2
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ISSN:0041-1345
1873-2623
DOI:10.1016/j.transproceed.2007.02.095