Molecular mechanisms exploited by Listeria monocytogenes during host cell invasion

The facultative intracellular bacterial pathogen Listeria monocytogenes has evolved multiple strategies to invade a large panel of mammalian cells. Host cell invasion is critical for several stages of listeriosis pathology such as the initial crossing of the host intestinal barrier and the successiv...

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Published inMicrobes and infection Vol. 9; no. 10; pp. 1167 - 1175
Main Authors Seveau, Stéphanie, Pizarro-Cerda, Javier, Cossart, Pascale
Format Journal Article
LanguageEnglish
Published France Elsevier SAS 01.08.2007
Elsevier
Subjects
Animals
Bacterial Proteins - metabolism
Bacterial Proteins - physiology
Female
Host tissue
Host-Pathogen Interactions
Humans
Life Sciences
Listeria monocytogenes
Listeria monocytogenes - metabolism
Listeria monocytogenes - pathogenicity
Listeriosis - microbiology
Mammalian cells
Microbiology and Parasitology
Nonphagocytic cells
Placenta
Placenta - microbiology
Pregnancy
Virulence Factors - metabolism
Virulence Factors - physiology
Listeria monocytogenes
Host tissue
Placenta
Nonphagocytic cells
Mammalian cells
LISTERIA MONOCYTOGENES
MAMMALIAN CELL
HOST TISSUE
NONPHAGOCYTIC CELL
PLACENTA
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Summary:The facultative intracellular bacterial pathogen Listeria monocytogenes has evolved multiple strategies to invade a large panel of mammalian cells. Host cell invasion is critical for several stages of listeriosis pathology such as the initial crossing of the host intestinal barrier and the successive colonization of diverse target organs including the placenta. In this review, we address the main molecular mechanisms known to be used by L. monocytogenes during invasion of nonphagocytic cells and host tissues.
Bibliography:ObjectType-Article-2
SourceType-Scholarly Journals-1
ObjectType-Feature-3
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ObjectType-Review-1
ISSN:1286-4579
1769-714X
DOI:10.1016/j.micinf.2007.05.004