Correction: Association of Genetic Variants with Isolated Fasting Hyperglycaemia and Isolated Postprandial Hyperglycaemia in a Han Chinese Population

Joint study of IPH associated SNPs, including TCF7L2, CDKAL1, KCNQ1, PRC1, TP53INP1 and GCKR, shows that individuals with IPH carry more risk alleles from the above loci than controls (ptrend < 0.0001; Figure 2A). In SNPs associated with IPH, the numbers of risk alleles carried in IFH subjects we...

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Published inPloS one Vol. 8; no. 8
Main Authors Kong, Xiaomu, Hong, Jing, Chen, Ying, Chen, Li, Zhao, Zhigang, Li, Qiang, Ge, Jiapu, Chen, Gang, Guo, Xiaohui, Lu, Juming, Weng, Jianping, Jia, Weiping, Ji, Linong, Xiao, Jianzhong, Shan, Zhongyan, Liu, Jie, Tian, Haoming, Ji, Qiuhe, Zhu, Dalong, Zhou, Zhiguang, Shan, Guangliang, Yang, Wenying
Format Journal Article
LanguageEnglish
Published United States Public Library of Science 2013
Public Library of Science (PLoS)
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Online AccessGet full text
ISSN1932-6203
1932-6203
DOI10.1371/annotation/936a4359-1bf5-4c33-be7d-1468e75eaa8b

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Abstract Joint study of IPH associated SNPs, including TCF7L2, CDKAL1, KCNQ1, PRC1, TP53INP1 and GCKR, shows that individuals with IPH carry more risk alleles from the above loci than controls (ptrend < 0.0001; Figure 2A). In SNPs associated with IPH, the numbers of risk alleles carried in IFH subjects were not significantly more than that in the control group (ptrend = 0.2752; Figure 2A); and the number of risk alleles did not increase the risk of IFH (1.053 [0.963-1.150] per allele, p = 0.2564; Figure 2C).
AbstractList [This corrects the article DOI: 10.1371/journal.pone.0071399.].[This corrects the article DOI: 10.1371/journal.pone.0071399.].
[This corrects the article DOI: 10.1371/journal.pone.0071399.].
Joint study of IPH associated SNPs, including TCF7L2, CDKAL1, KCNQ1, PRC1, TP53INP1 and GCKR, shows that individuals with IPH carry more risk alleles from the above loci than controls (ptrend < 0.0001; Figure 2A). In SNPs associated with IPH, the numbers of risk alleles carried in IFH subjects were not significantly more than that in the control group (ptrend = 0.2752; Figure 2A); and the number of risk alleles did not increase the risk of IFH (1.053 [0.963-1.150] per allele, p = 0.2564; Figure 2C).
Joint study of IPH associated SNPs, including TCF7L2, CDKAL1, KCNQ1, PRC1, TP53INP1 and GCKR, shows that individuals with IPH carry more risk alleles from the above loci than controls (ptrend < 0.0001; Figure 2A). In SNPs associated with IPH, the numbers of risk alleles carried in IFH subjects were not significantly more than that in the control group (ptrend = 0.2752; Figure 2A); and the number of risk alleles did not increase the risk of IFH (1.053 [0.963-1.150] per allele, p = 0.2564; Figure 2C).
Author Jia, Weiping
Yang, Wenying
Lu, Juming
Tian, Haoming
Zhu, Dalong
Chen, Ying
Shan, Guangliang
Chen, Li
Guo, Xiaohui
Liu, Jie
Ji, Qiuhe
Li, Qiang
Xiao, Jianzhong
Chen, Gang
Ge, Jiapu
Zhou, Zhiguang
Ji, Linong
Zhao, Zhigang
Weng, Jianping
Shan, Zhongyan
Hong, Jing
Kong, Xiaomu
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Snippet [This corrects the article DOI: 10.1371/journal.pone.0071399.].
Joint study of IPH associated SNPs, including TCF7L2, CDKAL1, KCNQ1, PRC1, TP53INP1 and GCKR, shows that individuals with IPH carry more risk alleles from the...
[This corrects the article DOI: 10.1371/journal.pone.0071399.].[This corrects the article DOI: 10.1371/journal.pone.0071399.].
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SubjectTerms Genetic diversity
Genetic variance
Hyperglycemia
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Title Correction: Association of Genetic Variants with Isolated Fasting Hyperglycaemia and Isolated Postprandial Hyperglycaemia in a Han Chinese Population
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https://www.proquest.com/docview/1974811254
https://www.proquest.com/docview/1984250807
https://pubmed.ncbi.nlm.nih.gov/PMC5754205
https://doaj.org/article/b1ef24c2e7d4498a9703b4350789db27
http://dx.doi.org/10.1371/annotation/936a4359-1bf5-4c33-be7d-1468e75eaa8b
Volume 8
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