Correction: Association of Genetic Variants with Isolated Fasting Hyperglycaemia and Isolated Postprandial Hyperglycaemia in a Han Chinese Population

Joint study of IPH associated SNPs, including TCF7L2, CDKAL1, KCNQ1, PRC1, TP53INP1 and GCKR, shows that individuals with IPH carry more risk alleles from the above loci than controls (ptrend < 0.0001; Figure 2A). In SNPs associated with IPH, the numbers of risk alleles carried in IFH subjects we...

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Published inPloS one Vol. 8; no. 8
Main Authors Kong, Xiaomu, Hong, Jing, Chen, Ying, Chen, Li, Zhao, Zhigang, Li, Qiang, Ge, Jiapu, Chen, Gang, Guo, Xiaohui, Lu, Juming, Weng, Jianping, Jia, Weiping, Ji, Linong, Xiao, Jianzhong, Shan, Zhongyan, Liu, Jie, Tian, Haoming, Ji, Qiuhe, Zhu, Dalong, Zhou, Zhiguang, Shan, Guangliang, Yang, Wenying
Format Journal Article
LanguageEnglish
Published United States Public Library of Science 2013
Public Library of Science (PLoS)
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Summary:Joint study of IPH associated SNPs, including TCF7L2, CDKAL1, KCNQ1, PRC1, TP53INP1 and GCKR, shows that individuals with IPH carry more risk alleles from the above loci than controls (ptrend < 0.0001; Figure 2A). In SNPs associated with IPH, the numbers of risk alleles carried in IFH subjects were not significantly more than that in the control group (ptrend = 0.2752; Figure 2A); and the number of risk alleles did not increase the risk of IFH (1.053 [0.963-1.150] per allele, p = 0.2564; Figure 2C).
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ISSN:1932-6203
1932-6203
DOI:10.1371/annotation/936a4359-1bf5-4c33-be7d-1468e75eaa8b