Correction: Single-cell deconvolution algorithms analysis unveils autocrine IL11-mediated resistance to docetaxel in prostate cancer via activation of the JAK1/STAT4 pathway
D Images of colony formation in prostate cancer cell lines PC3 and DU145, comparing the control and overexpression treated groups, alongside bar graphs quantifying the number of colonies formed, indicating a signifcant increase in colony formation upon IL-11 treatment. Bar graphs showing the percent...
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Published in | Journal of experimental & clinical cancer research Vol. 43; no. 1; pp. 333 - 4 |
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Main Authors | , , , , , , , , |
Format | Journal Article |
Language | English |
Published |
England
BioMed Central Ltd
28.12.2024
BioMed Central BMC |
Subjects | |
Online Access | Get full text |
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Summary: | D Images of colony formation in prostate cancer cell lines PC3 and DU145, comparing the control and overexpression treated groups, alongside bar graphs quantifying the number of colonies formed, indicating a signifcant increase in colony formation upon IL-11 treatment. Bar graphs showing the percentage of dead cells in prostate cancer cell lines PC3 (I) and DU145 (J), comparing the control and IL-11-treated groups. *P<0.05, **P<0.01, ***P<0.001 Full size image Correct Fig. 4 Fig. 4 figure 2 IL-11 contributes to increased viability and chemoresistance in prostate cancer cells. D Images of colony formation in prostate cancer cell lines PC3 and DU145, comparing the control and overexpression treated groups, alongside bar graphs quantifying the number of colonies formed, indicating a signifcant increase in colony formation upon IL-11 treatment. Bar graphs showing the percentage of dead cells in prostate cancer cell lines PC3 (I) and DU145 (J), comparing the control and IL-11-treated groups. *P<0.05, **P<0.01, ***P<0.001 Full size image The correction does not compromise the validity of the conclusions and the overall content of the article. |
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Bibliography: | correction ObjectType-Correction/Retraction-1 SourceType-Scholarly Journals-1 content type line 14 content type line 23 |
ISSN: | 1756-9966 0392-9078 1756-9966 |
DOI: | 10.1186/s13046-024-03257-8 |