Correction: Single-cell deconvolution algorithms analysis unveils autocrine IL11-mediated resistance to docetaxel in prostate cancer via activation of the JAK1/STAT4 pathway

D Images of colony formation in prostate cancer cell lines PC3 and DU145, comparing the control and overexpression treated groups, alongside bar graphs quantifying the number of colonies formed, indicating a signifcant increase in colony formation upon IL-11 treatment. Bar graphs showing the percent...

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Published inJournal of experimental & clinical cancer research Vol. 43; no. 1; pp. 333 - 4
Main Authors Cheng, Bisheng, Li, Lingfeng, Luo, Tianlong, Wang, Qiong, Luo, Yong, Bai, Shoumin, Li, Kaiwen, Lai, Yiming, Huang, Hai
Format Journal Article
LanguageEnglish
Published England BioMed Central Ltd 28.12.2024
BioMed Central
BMC
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Summary:D Images of colony formation in prostate cancer cell lines PC3 and DU145, comparing the control and overexpression treated groups, alongside bar graphs quantifying the number of colonies formed, indicating a signifcant increase in colony formation upon IL-11 treatment. Bar graphs showing the percentage of dead cells in prostate cancer cell lines PC3 (I) and DU145 (J), comparing the control and IL-11-treated groups. *P<0.05, **P<0.01, ***P<0.001 Full size image Correct Fig. 4 Fig. 4 figure 2 IL-11 contributes to increased viability and chemoresistance in prostate cancer cells. D Images of colony formation in prostate cancer cell lines PC3 and DU145, comparing the control and overexpression treated groups, alongside bar graphs quantifying the number of colonies formed, indicating a signifcant increase in colony formation upon IL-11 treatment. Bar graphs showing the percentage of dead cells in prostate cancer cell lines PC3 (I) and DU145 (J), comparing the control and IL-11-treated groups. *P<0.05, **P<0.01, ***P<0.001 Full size image The correction does not compromise the validity of the conclusions and the overall content of the article.
Bibliography:correction
ObjectType-Correction/Retraction-1
SourceType-Scholarly Journals-1
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ISSN:1756-9966
0392-9078
1756-9966
DOI:10.1186/s13046-024-03257-8