NLRC3 Restrains Responses to a T

The NOD-like receptor protein NLRC3 attenuates myeloid cell inflammatory responses. In this issue of Immunity, Uchimura et al. (2018) reveal additional T-cell-intrinsic functions for NLRC3 in restricting T cell metabolism, T helper 1 and T helper 17 cell responses, and antiviral and autoimmune respo...

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Published inImmunity (Cambridge, Mass.) Vol. 49; no. 6; pp. 989 - 991
Main Authors Holley, Caroline L., Coll, Rebecca C., Schroder, Kate
Format Journal Article
LanguageEnglish
Published United States Elsevier Inc 18.12.2018
Elsevier Limited
Subjects
Antigens
Autoimmune diseases
Autoimmunity
Carrier Proteins
Cytokines
Disease
Experiments
Immunity
Immunity, Innate
Infections
Inflammation
Intercellular Signaling Peptides and Proteins
Laboratories
Lymphocytes
Lymphocytes T
Metabolism
Proteins
Studies
T cell receptors
T-Lymphocytes
Tumor necrosis factor-TNF
Viral infections
Ireland
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Summary:The NOD-like receptor protein NLRC3 attenuates myeloid cell inflammatory responses. In this issue of Immunity, Uchimura et al. (2018) reveal additional T-cell-intrinsic functions for NLRC3 in restricting T cell metabolism, T helper 1 and T helper 17 cell responses, and antiviral and autoimmune responses. The NOD-like receptor protein NLRC3 attenuates myeloid cell inflammatory responses. In this issue of Immunity, Uchimura et al. (2018) reveal additional T-cell-intrinsic functions for NLRC3 in restricting T cell metabolism, T helper 1 and T helper 17 cell responses, and antiviral and autoimmune responses.
Bibliography:ObjectType-Article-2
SourceType-Scholarly Journals-1
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ObjectType-Commentary-1
ISSN:1074-7613
1097-4180
DOI:10.1016/j.immuni.2018.12.003