Spasticity and contracture. Physiologic aspects of formation

Disruption of the upper motor neuron inhibitory pathways by stroke, brain trauma, or spinal cord injury leads to muscle spasticity. Spasticity is characterized by increased muscle tone, hyperactive reflexes, and possible clonus or rigidity. The increased muscle tone may result in loss of joint motio...

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Bibliographic Details
Published inClinical orthopaedics and related research no. 233; p. 7
Main Authors Botte, M J, Nickel, V L, Akeson, W H
Format Journal Article
LanguageEnglish
Published United States 01.08.1988
Subjects
Animals
Contracture - physiopathology
Contracture - prevention & control
Contracture - therapy
Humans
Motor Neurons - physiology
Muscle Spasticity - physiopathology
Muscle Spasticity - prevention & control
Muscle Spasticity - therapy
Muscles - innervation
Neural Pathways - anatomy & histology
Neural Pathways - physiology
Neurons, Efferent - physiology
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Summary:Disruption of the upper motor neuron inhibitory pathways by stroke, brain trauma, or spinal cord injury leads to muscle spasticity. Spasticity is characterized by increased muscle tone, hyperactive reflexes, and possible clonus or rigidity. The increased muscle tone may result in loss of joint motion, leading to contractures. Treatment of established contractures is difficult. Prevention of contractures by joint mobilization is emphasized as a goal in the management of patients with spasticity.
ISSN:0009-921X
DOI:10.1097/00003086-198808000-00003