Extracellular matrix remodeling in heart failure : A role for de novo angiotensin II generation

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Bibliographic Details
Published inCirculation (New York, N.Y.) Vol. 96; no. 11; pp. 4065 - 4082
Main Author WEBER, K. T
Format Journal Article
LanguageEnglish
Published Hagerstown, MD Lippincott Williams & Wilkins 02.12.1997
American Heart Association, Inc
Subjects
Aged
Angiotensin II - biosynthesis
Angiotensin II - blood
Angiotensin-Converting Enzyme Inhibitors - therapeutic use
Animals
Biological and medical sciences
Cardiology. Vascular system
Collagen - metabolism
Coronary heart disease
Female
Fibrosis
Heart
Heart Failure - etiology
Heart Failure - pathology
Heart Failure - physiopathology
Humans
Medical sciences
Myocardial Infarction - complications
Myocardial Infarction - pathology
Myocardial Infarction - physiopathology
Myofibrils
Peptidyl-Dipeptidase A - drug effects
Peptidyl-Dipeptidase A - metabolism
Ventricular Dysfunction, Left - etiology
Ventricular Dysfunction, Left - pathology
Ventricular Dysfunction, Left - physiopathology
Human
Heart failure
Infarct
Rat
Pathogenesis
Rodentia
Cardiovascular disease
Coronary heart disease
Myocardial disease
Pathology
Vertebrata
Optical microscopy
Mammalia
Heart disease
Animal
Fibrosis
Myocardium
Complication
Extracellular matrix
Angiotensin II
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Bibliography:ObjectType-Case Study-3
SourceType-Scholarly Journals-1
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ObjectType-Review-1
ObjectType-Feature-5
ObjectType-Report-2
ObjectType-Article-4
ISSN:0009-7322
1524-4539
DOI:10.1161/01.CIR.96.11.4065