Impact of Long‐Wavelength Ultraviolet A1 and Visible Light on Light‐Skinned Individuals

Solar radiation is known to be a major contributor to the development of skin cancer. Most sunscreen formulations, including those with broad spectrum, offer minimal protection in long‐wavelength ultraviolet A1 (UVA1; 370–400 nm) and visible light (VL; 400–700 nm) domain. There is limited informatio...

Full description

Saved in:
Bibliographic Details
Published inPhotochemistry and photobiology Vol. 95; no. 6; pp. 1285 - 1287
Main Authors Kohli, Indermeet, Zubair, Raheel, Lyons, Alexis B., Nahhas, Amanda F., Braunberger, Taylor L., Mokhtari, Mohsen, Ruvolo, Eduardo, Lim, Henry W., Hamzavi, Iltefat H.
Format Journal Article
LanguageEnglish
Published United States Blackwell Publishing Ltd 01.11.2019
Subjects
Online AccessGet full text
ISSN0031-8655
1751-1097
1751-1097
DOI10.1111/php.13143

Cover

Loading…
More Information
Summary:Solar radiation is known to be a major contributor to the development of skin cancer. Most sunscreen formulations, including those with broad spectrum, offer minimal protection in long‐wavelength ultraviolet A1 (UVA1; 370–400 nm) and visible light (VL; 400–700 nm) domain. There is limited information regarding the impact of this broad waveband (VL + UVA1, 370–700 nm) on those with light skin. In this study, ten healthy adult subjects with Fitzpatrick skin phototypes I–III were enrolled. On day 0, subjects' lower back was exposed to a VL + UVA1 dose of 480 J cm−2. A statistically significant increase in erythema immediately after irradiation compared with subjects' baseline nonirradiated skin was observed. Clinically perceptible erythema with VL + UVA1 is a novel finding since the erythemogenic spectrum of sunlight has primarily been attributed to ultraviolet B and short‐wavelength ultraviolet A (320–340 nm). The results emphasize the need for protection against this part of the solar spectra and warrant further investigation. Solar radiation is known to be a major contributor to the development of skin cancer. Most sunscreen formulations, including those with broad spectrum, offer minimal protection in long wavelength ultraviolet A1 (UVA1; 370–400 nm) and visible light (VL; 400–700 nm) domain. There is limited information regarding the impact of this broad waveband (VL + UVA1, 370‐700 nm) on those with light skin. This study demonstrates that VL + UVA1 induces clinically perceptible erythema. This is a novel finding since the erythemogenic spectrum of sunlight has primarily been attributed to ultraviolet B and short wavelength ultraviolet A (320–340 nm).
Bibliography:SourceType-Scholarly Journals-1
ObjectType-Correspondence-2
content type line 14
ObjectType-Letter to the Editor-1
ObjectType-Article-2
ObjectType-Correspondence-1
content type line 23
ISSN:0031-8655
1751-1097
1751-1097
DOI:10.1111/php.13143