A reversible lentiform nucleus T1 hyperintensity in hypoglycemic chorea
Acute to subacute onset chorea is commonly due to stroke, metabolic, post-infectious, and autoimmune disorders. Chorea in diabetes (DM) patient is mostly due to hyperglycemia, and few cases were reported due to hypoglycemia. The mechanism at the cellular level is still evolving. The MRI changes in d...
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Published in | Annals of Movement Disorders Vol. 4; no. 1; pp. 39 - 41 |
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Main Authors | , , , |
Format | Journal Article |
Language | English |
Published |
Medknow Publications and Media Pvt. Ltd
01.01.2021
Wolters Kluwer Medknow Publications |
Subjects | |
Online Access | Get full text |
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Summary: | Acute to subacute onset chorea is commonly due to stroke, metabolic, post-infectious, and autoimmune disorders. Chorea in diabetes (DM) patient is mostly due to hyperglycemia, and few cases were reported due to hypoglycemia. The mechanism at the cellular level is still evolving. The MRI changes in diabetic chorea patients are variably reported. It is important to discuss our patient with hypoglycemic hemichorea because of unique features like low BMI, recently diagnosed DM on sulphonylurea with a history of hypoglycemic episodes, and acute left hemichorea that was controlled with neuroleptics, and her MRI brain showed reversible lentiform nucleus T1 hyperintensity without diffusion restriction and blooming on SWI. |
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ISSN: | 2590-3446 2590-3454 |
DOI: | 10.4103/AOMD.AOMD_40_20 |