Nonatherosclerotic causes of coronary artery narrowing—Part III

• Published in: Clinical cardiology (Mahwah, N.J.) Vol. 19; no. 8; pp. 656 - 661
• Main Authors: Waller, Bruce F., Fry, Edward T. A., Hermiller, James B., Peters, Thomas, Slack, John D.
• Format: Journal Article
• Language: English
• Published: New York Wiley Periodicals, Inc 01.08.1996; Wiley
• Subjects: Biological and medical sciences; Cardiology. Vascular system; Cocaine; Coronary Disease - etiology; Coronary heart disease; coronary vasculitis; Heart; Humans; Kawasaki's disease; Medical sciences; metastatic tumor; Mucocutaneous Lymph Node Syndrome - complications; Myocardial Infarction - etiology; Neoplasm Metastasis; Substance-Related Disorders - complications; Vasculitis - complications; Human; Kawasaki syndrome; Immunopathology; Drug addiction; Infarct; Coronary artery; Etiopathogenesis; Cardiovascular disease; Autoimmune disease; Coronary heart disease; Myocardial disease; Vascular disease; Vasculitis; Systemic disease; Myocardium; Tumor; Cocaine; Diagnosis; Takayashu arteritis
• ISSN: 0160-9289; 1932-8737
• DOI: 10.1002/clc.4960190814

Approximately 5% of patients with acute myocardial infarction do not have atherosclerotic coronary artery disease but have other causes for their luminal narrowing. The third part of this three‐part review of nonatherosclerotic causes of coronary narrowing focuses on coronary vasculitis, infectious diseases, Kawasaki's disease, metabolic disorders, metastatic disease, and substance abuse (cocaine). coronary arteritis: (1) focal artery necrosis with or without calcification, (2) acute coronary artery thrombosis or recanal‐ized thrombus unassociated with underlying atherosclerotic plaque, (3) rupture of the vessel wall unassociated with trauma or an interventional procedure, (4) coronary artery wall thickening with secondary luminal narrowing, and (5) wall thinning with aneurysm formation.3 Specific coronary lesions also may be seen in specific systemic diseases (e.g., pol‐yarteritis nodosa).