Molecular biology of scrapie-like agents

A detailed account is given of the nature of the causal agent of scrapie and other transmissible spongiform encephalopathies, with reference to proteinase-resistant protein and its gene, subviral particles and the prion hypothesis.

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Bibliographic Details
Published inRevue scientifique et technique (International Office of Epizootics) Vol. 11; no. 2; p. 569
Main Authors Bennett, A.D. (Agricultural and Food Research Council, Compton (Royaume Uni). Institute for Animal Health), Birkett, C.R, Bostock, C.J
Format Journal Article
LanguageEnglish
Published France 01.06.1992
Subjects
AETIOLOGY
AGENT PATHOGENE
Alleles
Amino Acid Sequence
ANIMAL DISEASES
Animals
Base Sequence
BIOLOGIA MOLECULAR
BIOLOGIE MOLECULAIRE
ENCEFALOPATIA ESPONGIFORME
ENCEPHALOPATHIE SPONGIFORME
ENFERMEDADES DE LOS ANIMALES
ETIOLOGIA
ETIOLOGIE
EXPRESION GENICA
EXPRESSION DES GENES
GENE EXPRESSION
Gene Expression Regulation, Viral
MALADIE DES ANIMAUX
MOLECULAR BIOLOGY
Molecular Sequence Data
ORGANISMOS PATOGENOS
PATHOGENS
Prions - biosynthesis
Prions - chemistry
Prions - genetics
PROTEINAS
PROTEINE
PROTEINS
PrPSc Proteins
RNA, Viral - chemistry
SCRAPIE AGENT
SPONGIFORM ENCEPHALOPATHY
VIRUS TEMBLADERA DE LOS OVINOS
VIRUS TREMBLANTE DU MOUTON
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Summary:A detailed account is given of the nature of the causal agent of scrapie and other transmissible spongiform encephalopathies, with reference to proteinase-resistant protein and its gene, subviral particles and the prion hypothesis.
Bibliography:L73
9400019
ISSN:0253-1933
DOI:10.20506/rst.11.2.610