The study of the somnological aspects of the human acute adaptation to the high-altitude

The primary cause of alteration in the neocortex electrogenesis in high-altitude adaptation at altitudes of 3000–5600 m above sea level, according to the EEG parameters of the night sleep and wakefulness, is uncompensated superficial hypoxia of the brain. The situation becomes worse at night due to...

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Bibliographic Details
Published inHuman physiology Vol. 40; no. 6; pp. 623 - 633
Main Authors Voynov, V. B., Verbitsky, E. V.
Format Journal Article
LanguageEnglish
Published Moscow Pleiades Publishing 04.12.2014
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Summary:The primary cause of alteration in the neocortex electrogenesis in high-altitude adaptation at altitudes of 3000–5600 m above sea level, according to the EEG parameters of the night sleep and wakefulness, is uncompensated superficial hypoxia of the brain. The situation becomes worse at night due to apnea/hypopnea effects during sleep, which occur as a result of muscular atony during slow falling asleep. The compensation of these disorders via hypertensive and cardiorespiratory responses is determined by general mechanisms and individual strategy.
ISSN:0362-1197
1608-3164
DOI:10.1134/S036211971405017X