Chronic treatment with amyloid β1-42 inhibits non-cholinergic high-affinity choline transport in NG108-15 cells through protein kinase C signaling

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Bibliographic Details
Published inBrain research Vol. 1062; no. 1-2; pp. 101 - 110
Main Authors NOVAKOVA, Jana, MIKASOVA, Lenka, MACHOVA, Eva, LISA, Vera, DOLEZAL, Vladimir
Format Journal Article
LanguageEnglish
Published London Elsevier 16.11.2005
Amsterdam
New York, NY
Subjects
Biological and medical sciences
Degenerative and inherited degenerative diseases of the nervous system. Leukodystrophies. Prion diseases
Medical sciences
Neurology
High-affinity choline transport
Protein kinase C
Rat
Alzheimer disease
Pathogenesis
Biological transport
Hemicholinium-3 binding
Signal transduction
Degenerative disease
Carrier protein
β-amyloid
Choline transporter-like protein
Nervous system diseases
Hybrid cell
Enzyme
Transferases
Rodentia
Cerebral disorder
Vertebrata
Chronic
Choline transporter 1
Mammalia
Treatment
Mouse
β Amyloid protein
Animal
Choline
Central nervous system disease
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ISSN:0006-8993
1872-6240
DOI:10.1016/j.brainres.2005.09.021