Isolated hypoglossal nerve palsy due to internal carotid artery dissection
Antiplatelet and anticoagulant treatments appear equally effective. 2 The more common manifestations of internal carotid artery dissection are headache, neck pain, ipsilateral oculosympathetic paresis (Horner's syndrome), and transient cerebral and ocular ischaemia. 1 The hypoglossal nerve orig...
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Published in | Practical neurology Vol. 17; no. 3; pp. 233 - 234 |
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Main Authors | , |
Format | Journal Article |
Language | English |
Published |
England
BMJ Publishing Group LTD
01.06.2017
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Subjects | |
Online Access | Get full text |
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Summary: | Antiplatelet and anticoagulant treatments appear equally effective. 2 The more common manifestations of internal carotid artery dissection are headache, neck pain, ipsilateral oculosympathetic paresis (Horner's syndrome), and transient cerebral and ocular ischaemia. 1 The hypoglossal nerve originates from the hypoglossal (XIIth cranial nerve) nucleus in the medial medulla, exits the skull via the hypoglossal canal, and passes between the internal jugular vein and internal carotid artery at the skull base. 3 At this point, it is vulnerable to compression from a high internal carotid artery dissection-mural thrombus causes eccentric expansion of the arterial wall-compressing the nerve. 4 The cervical sympathetic trunk and cranial nerves IX, X and XI also lie in close proximity to the internal carotid artery at this level; a dissection can also compress these ( figure 2 ). |
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Bibliography: | SourceType-Other Sources-1 content type line 63 ObjectType-Editorial-2 ObjectType-Commentary-1 |
ISSN: | 1474-7758 1474-7766 |
DOI: | 10.1136/practneurol-2017-001617 |