Hepatic stellate cells-specific LOXL1 deficiency abrogates hepatic inflammation, fibrosis, and corrects lipid metabolic abnormalities in non-obese NASH mice - Yang, Aiting; Yan, Xuzhen; Fan, Xu; Shi, Yiwen; Huang, Tao; Li, Weiyu; Chen, Wei; Jia, Jidong; You, Hong (Hepatology international, 2021)
Keywords: Abnormalities; Adipose tissue; Amino Acid Oxidoreductases - genetics; Amino acids; Animal models; Animals; Biopsy; Choline; Colorectal Surgery; Crosslinking; Diet; Disease Models, Animal; Extracellular matrix; Fatty liver; Fibrosis; Gene expression; Hepatic Stellate Cells - pathology; Hepatology; Humans; Inflammation; Inflammation - pathology; Leptin; Lipids; Lipogenesis; Liver; Liver - pathology; Liver Cirrhosis - pathology; Liver diseases; Lysyl oxidase; Medicine; Medicine & Public Health; Metabolic disorders; Metabolism; Mice; Mice, Inbred C57BL; Non-alcoholic Fatty Liver Disease - genetics; Non-alcoholic Fatty Liver Disease - pathology; Nutrient deficiency; Obesity; Original Article; Pathogenesis; Steatosis; Stellate cells; Surgery; NAFLD; Leptin; Liver fibrosis; Hepatic stellate cell; Lipid metabolism; Crosslink; ECM; LOXL1; Non-obese NASH; CDAA