Hemodynamic changes of left gastric vein before and after endoscropic scleroligation for esophageal varices
We investigated hemodynamic changes following endoscopic sclero-ligation therapy (ESL : EIS combined with EVL therapy) which was performed in six patients with esophageal varices due to liver cirrhosis. Angioraphy before ESL demonstrated that both of the left gastric artery (LGA) and the left gastri...
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Published in | Japanese Journal of Portal Hypertension and Esophageal Varices Vol. 3; no. 3; pp. 201 - 207 |
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Main Authors | , , , , , , , , , , , , , , , , |
Format | Journal Article |
Language | Japanese |
Published |
The Japan Society for Portal Hypertension
10.12.1997
日本門脈圧亢進症学会 |
Subjects | |
Online Access | Get full text |
ISSN | 1341-6375 2187-1213 |
DOI | 10.11423/jsph1995.3.3_201 |
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Summary: | We investigated hemodynamic changes following endoscopic sclero-ligation therapy (ESL : EIS combined with EVL therapy) which was performed in six patients with esophageal varices due to liver cirrhosis. Angioraphy before ESL demonstrated that both of the left gastric artery (LGA) and the left gastric vein (LGV) were the feeding vessels for esophageal varices in five patients. Varices in the other patient was fed solely by LGA. Before ESL, flow direction of LGV was hepatoufugal in four, and “to and fro” in two. After ESL, esophageal varices were eradicated in five patients, endoscopically. Of these, flow direction of LGV changed from hepatofugal to hepatopetal in two, from hepatofugal to “to and fro” in two. And in one patient with “to and fro”, the flow of LGV was not demonstrated. The diameter of LGV decreased in three out of four patients with patent LGV after ESL. Esophageal varices were not eradicated in one patient whose varices were fed by LGA alone. ESL neither changed the flow direction nor the diameter of the LGV. It is suggested that ESL embolized LGV completely or imcompletely, and caused subsequent hemodynamic changes for patients for whom LGV was the feeder vessel to the varices. |
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ISSN: | 1341-6375 2187-1213 |
DOI: | 10.11423/jsph1995.3.3_201 |