Parkinson's disease–associated mutant VPS35 causes mitochondrial dysfunction by recycling DLP1 complexes - Wang, Wenzhang; Wang, Xinglong; Fujioka, Hisashi; Hoppel, Charles; Whone, Alan L; Caldwell, Maeve A; Cullen, Peter J; Liu, Jun; Zhu, Xiongwei (Nature medicine, 2016)
Keywords: 13/1; 13/106; 13/109; 13/51; 13/89; 14/1; 14/19; 14/28; 14/33; 14/35; 38/111; 38/70; 631/378/1689/1718; 631/378/87; 64/60; 82/29; Aged; Aged, 80 and over; Animals; Biomedicine; Blotting, Western; Cancer Research; Cell Line, Tumor; Development and progression; Dynamins - metabolism; Female; Fluorescence Recovery After Photobleaching; Fluorescent Antibody Technique; Gene Knockdown Techniques; Gene mutations; Genetic aspects; GTP Phosphohydrolases - metabolism; Humans; In Vitro Techniques; Infectious Diseases; Male; Metabolic Diseases; Mice; Microscopy, Electron, Transmission; Microscopy, Immunoelectron; Microtubule-Associated Proteins - metabolism; Mitochondria; Mitochondria - metabolism; Mitochondria - pathology; Mitochondrial diseases; Mitochondrial DNA; Mitochondrial Dynamics - genetics; Mitochondrial Proteins - metabolism; Molecular Medicine; Mutation; Neurology; Neurons; Neurosciences; Oxidative Stress; Parkinson Disease - genetics; Parkinson Disease - metabolism; Parkinson Disease - pathology; Parkinson's disease; Pathogenesis; Physiological aspects; Quality control; Rats; Risk factors; Substantia Nigra - cytology; Time-Lapse Imaging; Vesicular Transport Proteins - genetics; United States