A short-term intervention trial with selenate, selenium-enriched yeast and selenium-enriched milk: effects on oxidative defence regulation
Increased Se intakes have been associated with decreased risk of cancer and CVD. Several mechanisms have been proposed, including antioxidant effects through selenoproteins, induction of carcinogen metabolism and effects on the blood lipid profile. In a 4 × 1 week randomised, double-blind cross-over...
Saved in:
| Published in | British journal of nutrition Vol. 99; no. 4; pp. 883 - 892 |
|---|---|
| Main Authors | , , , , , , , , |
| Format | Journal Article |
| Language | English |
| Published |
Cambridge, UK
Cambridge University Press
01.04.2008
|
| Subjects | |
| Online Access | Get full text |
Cover
Loading…
| Abstract | Increased Se intakes have been associated with decreased risk of cancer and CVD. Several mechanisms have been proposed, including antioxidant effects through selenoproteins, induction of carcinogen metabolism and effects on the blood lipid profile. In a 4 × 1 week randomised, double-blind cross-over study, healthy young men supplemented their usual diet with selenate, Se-enriched yeast, Se-enriched milk or placebo (Se dose was 300 μg/d for selenate and Se-enriched yeast, and about 480 μg/d for Se-enriched milk) followed by 8-week washout periods. All Se sources increased serum Se levels after supplementation for 1 week. The effect of the organic forms did not differ significantly and both increased serum Se more than selenate. Conversely, thrombocyte glutathione peroxidase (GPX) was increased in the periods where subjects were supplemented with selenate but not in those where they were given Se-enriched yeast or Se-enriched milk. We found no effect on plasma lipid resistance to oxidation, total cholesterol, TAG, HDL- and LDL-cholesterol, GPX, glutathione reductase (GR) and glutathione S-transferase (GST) activities measured in erythrocytes, GPX and GR activities determined in plasma, or GR and GST activities in thrombocytes. Leucocyte expression of genes encoding selenoproteins (GPX1, TrR1 and SelP), and of electrophile response element-regulated genes (GCLC, Fra1 and NQO1) were likewise unaffected at all time points following intervention. We conclude that thrombocyte GPX is specifically increased by short-term selenate supplementation, but not by short-term supplementation with organic Se. Short-term Se supplementation does not seem to affect blood lipid markers or expression and activity of selected enzymes and a transcription factor involved in glutathione-mediated detoxification and antioxidation. |
|---|---|
| AbstractList | Increased Se intakes have been associated with decreased risk of cancer and CVD. Several mechanisms have been proposed, including antioxidant effects through selenoproteins, induction of carcinogen metabolism and effects on the blood lipid profile. In a 4 x 1 week randomised, double-blind cross-over study, healthy young men supplemented their usual diet with selenate, Se-enriched yeast, Se-enriched milk or placebo (Se dose was 300 μg/d for selenate and Se-enriched yeast, and about 480 μg/d for Se-enriched milk) followed by 8-week washout periods. All Se sources increased serum Se levels after supplementation for 1 week. The effect of the organic forms did not differ significantly and both increased serum Se more than selenate. Conversely, thrombocyte glutathione peroxidase (GPX) was increased in the periods where subjects were supplemented with selenate but not in those where they were given Se-enriched yeast or Se-enriched milk. We found no effect on plasma lipid resistance to oxidation, total cholesterol, TAG, HDL- and LDL-cholesterol, GPX, glutathione reductase (GR) and glutathione S-transferase (GST) activities measured in erythrocytes, GPX and GR activities determined in plasma, or GR and GST activities in thrombocytes. Leucocyte expression of genes encoding selenoproteins (GPX1, TrR1 and SelP), and of electrophile response element-regulated genes (GCLC, Fra1 and NQO1) were likewise unaffected at all time points following intervention. We conclude that thrombocyte GPX is specifically increased by short-term selenate supplementation, but not by short-term supplementation with organic Se. Short-term Se supplementation does not seem to affect blood lipid markers or expression and activity of selected enzymes and a transcription factor involved in glutathione-mediated detoxification and antioxidation. Increased Se intakes have been associated with decreased risk of cancer and CVD. Several mechanisms have been proposed, including antioxidant effects through selenoproteins, induction of carcinogen metabolism and effects on the blood lipid profile. In a 4 x 1 week randomised, double-blind cross-over study, healthy young men supplemented their usual diet with selenate, Se-enriched yeast, Se-enriched milk or placebo (Se dose was 300 microg/d for selenate and Se-enriched yeast, and about 480 microg/d for Se-enriched milk) followed by 8-week washout periods. All Se sources increased serum Se levels after supplementation for 1 week. The effect of the organic forms did not differ significantly and both increased serum Se more than selenate. Conversely, thrombocyte glutathione peroxidase (GPX) was increased in the periods where subjects were supplemented with selenate but not in those where they were given Se-enriched yeast or Se-enriched milk. We found no effect on plasma lipid resistance to oxidation, total cholesterol, TAG, HDL- and LDL-cholesterol, GPX, glutathione reductase (GR) and glutathione S-transferase (GST) activities measured in erythrocytes, GPX and GR activities determined in plasma, or GR and GST activities in thrombocytes. Leucocyte expression of genes encoding selenoproteins (GPX1, TrR1 and SelP), and of electrophile response element-regulated genes (GCLC, Fra1 and NQO1) were likewise unaffected at all time points following intervention. We conclude that thrombocyte GPX is specifically increased by short-term selenate supplementation, but not by short-term supplementation with organic Se. Short-term Se supplementation does not seem to affect blood lipid markers or expression and activity of selected enzymes and a transcription factor involved in glutathione-mediated detoxification and antioxidation. Increased Se intakes have been associated with decreased risk of cancer and CVD. Several mechanisms have been proposed, including antioxidant effects through selenoproteins, induction of carcinogen metabolism and effects on the blood lipid profile. In a 4 x 1 week randomised, double-blind cross-over study, healthy young men supplemented their usual diet with selenate, Se-enriched yeast, Se-enriched milk or placebo (Se dose was 300 microg/d for selenate and Se-enriched yeast, and about 480 microg/d for Se-enriched milk) followed by 8-week washout periods. All Se sources increased serum Se levels after supplementation for 1 week. The effect of the organic forms did not differ significantly and both increased serum Se more than selenate. Conversely, thrombocyte glutathione peroxidase (GPX) was increased in the periods where subjects were supplemented with selenate but not in those where they were given Se-enriched yeast or Se-enriched milk. We found no effect on plasma lipid resistance to oxidation, total cholesterol, TAG, HDL- and LDL-cholesterol, GPX, glutathione reductase (GR) and glutathione S-transferase (GST) activities measured in erythrocytes, GPX and GR activities determined in plasma, or GR and GST activities in thrombocytes. Leucocyte expression of genes encoding selenoproteins (GPX1, TrR1 and SelP), and of electrophile response element-regulated genes (GCLC, Fra1 and NQO1) were likewise unaffected at all time points following intervention. We conclude that thrombocyte GPX is specifically increased by short-term selenate supplementation, but not by short-term supplementation with organic Se. Short-term Se supplementation does not seem to affect blood lipid markers or expression and activity of selected enzymes and a transcription factor involved in glutathione-mediated detoxification and antioxidation.Increased Se intakes have been associated with decreased risk of cancer and CVD. Several mechanisms have been proposed, including antioxidant effects through selenoproteins, induction of carcinogen metabolism and effects on the blood lipid profile. In a 4 x 1 week randomised, double-blind cross-over study, healthy young men supplemented their usual diet with selenate, Se-enriched yeast, Se-enriched milk or placebo (Se dose was 300 microg/d for selenate and Se-enriched yeast, and about 480 microg/d for Se-enriched milk) followed by 8-week washout periods. All Se sources increased serum Se levels after supplementation for 1 week. The effect of the organic forms did not differ significantly and both increased serum Se more than selenate. Conversely, thrombocyte glutathione peroxidase (GPX) was increased in the periods where subjects were supplemented with selenate but not in those where they were given Se-enriched yeast or Se-enriched milk. We found no effect on plasma lipid resistance to oxidation, total cholesterol, TAG, HDL- and LDL-cholesterol, GPX, glutathione reductase (GR) and glutathione S-transferase (GST) activities measured in erythrocytes, GPX and GR activities determined in plasma, or GR and GST activities in thrombocytes. Leucocyte expression of genes encoding selenoproteins (GPX1, TrR1 and SelP), and of electrophile response element-regulated genes (GCLC, Fra1 and NQO1) were likewise unaffected at all time points following intervention. We conclude that thrombocyte GPX is specifically increased by short-term selenate supplementation, but not by short-term supplementation with organic Se. Short-term Se supplementation does not seem to affect blood lipid markers or expression and activity of selected enzymes and a transcription factor involved in glutathione-mediated detoxification and antioxidation. Increased Se intakes have been associated with decreased risk of cancer and CVD. Several mechanisms have been proposed, including antioxidant effects through selenoproteins, induction of carcinogen metabolism and effects on the blood lipid profile. In a 4 x 1 week randomised, double-blind cross-over study, healthy young men supplemented their usual diet with selenate, Se-enriched yeast, Se-enriched milk or placebo (Se dose was 300 mu g/d for selenate and Se-enriched yeast, and about 480 mu g/d for Se-enriched milk) followed by 8-week washout periods. All Se sources increased serum Se levels after supplementation for 1 week. The effect of the organic forms did not differ significantly and both increased serum Se more than selenate. Conversely, thrombocyte glutathione peroxidase (GPX) was increased in the periods where subjects were supplemented with selenate but not in those where they were given Se-enriched yeast or Se-enriched milk. We found no effect on plasma lipid resistance to oxidation, total cholesterol, TAG, HDL- and LDL-cholesterol, GPX, glutathione reductase (GR) and glutathione S-transferase (GST) activities measured in erythrocytes, GPX and GR activities determined in plasma, or GR and GST activities in thrombocytes. Leucocyte expression of genes encoding selenoproteins (GPX1, TrR1 and SelP), and of electrophile response element-regulated genes (GCLC, Fra1 and NQO1) were likewise unaffected at all time points following intervention. We conclude that thrombocyte GPX is specifically increased by short-term selenate supplementation, but not by short-term supplementation with organic Se. Short-term Se supplementation does not seem to affect blood lipid markers or expression and activity of selected enzymes and a transcription factor involved in glutathione-mediated detoxification and antioxidation. Increased Se intakes have been associated with decreased risk of cancer and CVD. Several mechanisms have been proposed, including antioxidant effects through selenoproteins, induction of carcinogen metabolism and effects on the blood lipid profile. In a 4 1 week randomised, double-blind cross-over study, healthy young men supplemented their usual diet with selenate, Se-enriched yeast, Se-enriched milk or placebo (Se dose was 300 mu g/d for selenate and Se-enriched yeast, and about 480 mu g/d for Se-enriched milk) followed by 8-week washout periods. All Se sources increased serum Se levels after supplementation for 1 week. The effect of the organic forms did not differ significantly and both increased serum Se more than selenate. Conversely, thrombocyte glutathione peroxidase (GPX) was increased in the periods where subjects were supplemented with selenate but not in those where they were given Se-enriched yeast or Se-enriched milk. We found no effect on plasma lipid resistance to oxidation, total cholesterol, TAG, HDL- and LDL-cholesterol, GPX, glutathione reductase (GR) and glutathione S-transferase (GST) activities measured in erythrocytes, GPX and GR activities determined in plasma, or GR and GST activities in thrombocytes. Leucocyte expression of genes encoding selenoproteins (GPX1, TrR1 and SelP), and of electrophile response element-regulated genes (GCLC, Fra1 and NQO1) were likewise unaffected at all time points following intervention. We conclude that thrombocyte GPX is specifically increased by short-term selenate supplementation, but not by short-term supplementation with organic Se. Short-term Se supplementation does not seem to affect blood lipid markers or expression and activity of selected enzymes and a transcription factor involved in glutathione-mediated detoxification and antioxidation. Increased Se intakes have been associated with decreased risk of cancer and CVD. Several mechanisms have been proposed, including antioxidant effects through selenoproteins, induction of carcinogen metabolism and effects on the blood lipid profile. In a 4 x 1 week randomised, double-blind cross-over study, healthy young men supplemented their usual diet with selenate, Se-enriched yeast, Se-enriched milk or placebo (Se dose was 300 μg/d for selenate and Se-enriched yeast, and about 480 μg/d for Se-enriched milk) followed by 8-week washout periods. All Se sources increased serum Se levels after supplementation for 1 week. The effect of the organic forms did not differ significantly and both increased serum Se more than selenate. Conversely, thrombocyte glutathione peroxidase (GPX) was increased in the periods where subjects were supplemented with selenate but not in those where they were given Se-enriched yeast or Se-enriched milk. We found no effect on plasma lipid resistance to oxidation, total cholesterol, TAG, HDL- and LDL-cholesterol, GPX, glutathione reductase (GR) and glutathione S-transferase (GST) activities measured in erythrocytes, GPX and GR activities determined in plasma, or GR and GST activities in thrombocytes. Leucocyte expression of genes encoding selenoproteins (GPX1, TrR1 and SelP), and of electrophile response element-regulated genes (GCLC, Fra1 and NQO1) were likewise unaffected at all time points following intervention. We conclude that thrombocyte GPX is specifically increased by short-term selenate supplementation, but not by short-term supplementation with organic Se. Short-term Se supplementation does not seem to affect blood lipid markers or expression and activity of selected enzymes and a transcription factor involved in glutathione-mediated detoxification and antioxidation. [PUBLICATION ABSTRACT] Increased Se intakes have been associated with decreased risk of cancer and CVD. Several mechanisms have been proposed, including antioxidant effects through selenoproteins, induction of carcinogen metabolism and effects on the blood lipid profile. In a 4 × 1 week randomised, double-blind cross-over study, healthy young men supplemented their usual diet with selenate, Se-enriched yeast, Se-enriched milk or placebo (Se dose was 300 μg/d for selenate and Se-enriched yeast, and about 480 μg/d for Se-enriched milk) followed by 8-week washout periods. All Se sources increased serum Se levels after supplementation for 1 week. The effect of the organic forms did not differ significantly and both increased serum Se more than selenate. Conversely, thrombocyte glutathione peroxidase (GPX) was increased in the periods where subjects were supplemented with selenate but not in those where they were given Se-enriched yeast or Se-enriched milk. We found no effect on plasma lipid resistance to oxidation, total cholesterol, TAG, HDL- and LDL-cholesterol, GPX, glutathione reductase (GR) and glutathione S-transferase (GST) activities measured in erythrocytes, GPX and GR activities determined in plasma, or GR and GST activities in thrombocytes. Leucocyte expression of genes encoding selenoproteins (GPX1, TrR1 and SelP), and of electrophile response element-regulated genes (GCLC, Fra1 and NQO1) were likewise unaffected at all time points following intervention. We conclude that thrombocyte GPX is specifically increased by short-term selenate supplementation, but not by short-term supplementation with organic Se. Short-term Se supplementation does not seem to affect blood lipid markers or expression and activity of selected enzymes and a transcription factor involved in glutathione-mediated detoxification and antioxidation. Increased Se intakes have been associated with decreased risk of cancer and CVD. Several mechanisms have been proposed, including antioxidant effects through selenoproteins, induction of carcinogen metabolism and effects on the blood lipid profile. In a 4 × 1 week randomised, double-blind cross-over study, healthy young men supplemented their usual diet with selenate, Se-enriched yeast, Se-enriched milk or placebo (Se dose was 300 μg/d for selenate and Se-enriched yeast, and about 480 μg/d for Se-enriched milk) followed by 8-week washout periods. All Se sources increased serum Se levels after supplementation for 1 week. The effect of the organic forms did not differ significantly and both increased serum Se more than selenate. Conversely, thrombocyte glutathione peroxidase (GPX) was increased in the periods where subjects were supplemented with selenate but not in those where they were given Se-enriched yeast or Se-enriched milk. We found no effect on plasma lipid resistance to oxidation, total cholesterol, TAG, HDL- and LDL-cholesterol, GPX, glutathione reductase (GR) and glutathione S-transferase (GST) activities measured in erythrocytes, GPX and GR activities determined in plasma, or GR and GST activities in thrombocytes. Leucocyte expression of genes encoding selenoproteins ( GPX1 , TrR1 and SelP ), and of electrophile response element-regulated genes ( GCLC , Fra1 and NQO1 ) were likewise unaffected at all time points following intervention. We conclude that thrombocyte GPX is specifically increased by short-term selenate supplementation, but not by short-term supplementation with organic Se. Short-term Se supplementation does not seem to affect blood lipid markers or expression and activity of selected enzymes and a transcription factor involved in glutathione-mediated detoxification and antioxidation. |
| Author | Larsen, Erik H. Bügel, Susanne Ravn-Haren, Gitte Hoac, Tien Jørgensen, Karina Stagsted, Jan Bresson, June R. Krath, Britta N. Dragsted, Lars O. |
| Author_xml | – sequence: 1 givenname: Gitte surname: Ravn-Haren fullname: Ravn-Haren, Gitte email: grh@food.dtu.dk organization: Department of Toxicology and Risk Assessment, National Food Institute, Technical University of Denmark, Mørkhøj Bygade 19, 2860 Søborg, Denmark – sequence: 2 givenname: Susanne surname: Bügel fullname: Bügel, Susanne organization: Department of Human Nutrition and Centre for Advanced Food Studies, Faculty of Life Sciences, University of Copenhagen, Frederiksberg, Denmark – sequence: 3 givenname: Britta N. surname: Krath fullname: Krath, Britta N. organization: Department of Toxicology and Risk Assessment, National Food Institute, Technical University of Denmark, Mørkhøj Bygade 19, 2860 Søborg, Denmark – sequence: 4 givenname: Tien surname: Hoac fullname: Hoac, Tien organization: Biomedical Nutrition, Lund Institute of Technology, Lund University, Lund, Sweden – sequence: 5 givenname: Jan surname: Stagsted fullname: Stagsted, Jan organization: Institute of Food Science, Research Centre Foulum, Faculty of Agricultural Sciences, University of Aarhus, Tjele, Denmark – sequence: 6 givenname: Karina surname: Jørgensen fullname: Jørgensen, Karina organization: Department of Human Nutrition and Centre for Advanced Food Studies, Faculty of Life Sciences, University of Copenhagen, Frederiksberg, Denmark – sequence: 7 givenname: June R. surname: Bresson fullname: Bresson, June R. organization: Department of Toxicology and Risk Assessment, National Food Institute, Technical University of Denmark, Mørkhøj Bygade 19, 2860 Søborg, Denmark – sequence: 8 givenname: Erik H. surname: Larsen fullname: Larsen, Erik H. organization: Department of Food Chemistry, National Food Institute, Technical University of Denmark, Søborg, Denmark – sequence: 9 givenname: Lars O. surname: Dragsted fullname: Dragsted, Lars O. organization: Department of Toxicology and Risk Assessment, National Food Institute, Technical University of Denmark, Mørkhøj Bygade 19, 2860 Søborg, Denmark |
| BackLink | http://pascal-francis.inist.fr/vibad/index.php?action=getRecordDetail&idt=20214781$$DView record in Pascal Francis https://www.ncbi.nlm.nih.gov/pubmed/17888202$$D View this record in MEDLINE/PubMed https://lup.lub.lu.se/record/1206490$$DView record from Swedish Publication Index oai:portal.research.lu.se:publications/44def329-ed09-43c4-b2ab-8f349fd4617e$$DView record from Swedish Publication Index |
| BookMark | eNqNkstu1TAQhiNURC_wAGwgQoIVAdtx7IRdVdECqoRQKYturIk96XHrJKe208sr8NT49BxaqVDKInKs-eYfz8y_ma0N44BZ9pySd5RQ-f6AECIp5RWRNatoVT7KNiiXVcGEYGvZxiJcLOLr2WYIJ-laU9I8ydaprOuaEbaR_dzOw2z0sYjo-9wO6TjHIdpxyKO34PILG2d5QIcDRHy7_LNTX-DgrZ6hya8QQsxhMH-J9dadfsix61DHkCfN8dIaiPYcc4MdDhpzj8eTg0XBp9njDlzAZ6tzKzvc_fh951Ox_3Xv8872fqElE7EwjIMETA1AJREbISphZEU4MFOVptKsEVDKCstOtJwbEDyRVUepqEU6yq0MlrrhAudTq-be9uCv1AhWzdMowCmPAcHrmXKTCqgS5ay-fmRQSRK7kjUKDWkULzVXLYNW1V3Jm85wQSWmGvv31nDTPH3tSvs_5d4s5eZ-PJswRNXboNE5GHCcgpKklKRi7EGQS5lMQOSDIG0axmtWJ_DVHfBknPyQFqQYLWvW8IYk6MUKmtoezU27v32WgNcrAIIG13kYtA03XEKSb-vFbuiS034MwWN3K0XUwvPqD8-nHHknR9t4vazowbp_ZhbLTBsiXt6UAn-qhEwWUmLvm_qxd8Tl0e4XtZP4l0u-g1HBsU8dHB4wQss007pKRkxEuXoL9K235hhvx3X_O34Bp9otdg |
| CODEN | BJNUAV |
| CitedBy_id | crossref_primary_10_1016_j_jtemb_2022_127046 crossref_primary_10_1016_j_obmed_2019_100113 crossref_primary_10_1016_j_aca_2009_08_013 crossref_primary_10_1017_S0954422417000191 crossref_primary_10_1186_s12263_018_0608_4 crossref_primary_10_1007_s12263_011_0246_6 crossref_primary_10_1080_09637486_2016_1205550 crossref_primary_10_1016_j_yjmcc_2015_07_005 crossref_primary_10_1038_ejcn_2015_78 crossref_primary_10_1016_j_prostaglandins_2024_106901 crossref_primary_10_1007_s11224_011_9816_x crossref_primary_10_5625_lar_2011_27_1_9 crossref_primary_10_1007_s42000_019_00143_3 crossref_primary_10_1017_S0007114511005484 crossref_primary_10_1007_s12011_009_8588_2 crossref_primary_10_5806_AST_2014_27_2_92 crossref_primary_10_1155_2022_4715965 crossref_primary_10_1007_s40200_021_00821_3 crossref_primary_10_1016_j_numecd_2009_12_014 crossref_primary_10_1016_j_obmed_2020_100182 crossref_primary_10_1016_j_jtemb_2021_126801 crossref_primary_10_3390_nu6104002 crossref_primary_10_1002_14651858_CD009671_pub2 crossref_primary_10_1002_14651858_CD009673 crossref_primary_10_1002_ejlt_201200190 crossref_primary_10_1002_mnfr_200700369 crossref_primary_10_1007_s12011_010_8831_x crossref_primary_10_1515_CCLM_2009_261 crossref_primary_10_3389_fnut_2022_962312 crossref_primary_10_1016_j_ejmech_2018_07_063 crossref_primary_10_1016_j_ijheh_2014_04_005 crossref_primary_10_1002_ardp_201000014 crossref_primary_10_1016_j_jtemb_2019_126435 crossref_primary_10_1042_BCJ20170920 crossref_primary_10_1016_j_bcp_2011_06_025 crossref_primary_10_3390_nu6031251 crossref_primary_10_1007_s00394_015_1118_4 crossref_primary_10_1080_10590501_2015_1055163 crossref_primary_10_1017_S0007114515003499 crossref_primary_10_1080_10408398_2024_2402998 crossref_primary_10_3390_nu9070739 crossref_primary_10_1016_j_jtemb_2023_127273 crossref_primary_10_1017_S0022029908003324 crossref_primary_10_1016_j_jtemb_2023_127199 crossref_primary_10_1080_10408398_2017_1406332 crossref_primary_10_1017_S1446545000000397 crossref_primary_10_1039_c2mt00163b crossref_primary_10_3390_antiox10020167 crossref_primary_10_1016_j_jtemb_2018_02_024 crossref_primary_10_1016_j_clnu_2018_03_011 crossref_primary_10_1002_14651858_CD007176_pub2 crossref_primary_10_1093_nutrit_nuaa136 crossref_primary_10_1016_j_jtemb_2014_11_008 crossref_primary_10_1042_BSR20090089 crossref_primary_10_1021_jf201501x crossref_primary_10_1002_14651858_CD005195_pub4 crossref_primary_10_1002_em_20490 crossref_primary_10_1007_s00253_019_09922_9 crossref_primary_10_1002_pros_21012 |
| Cites_doi | 10.1093/ajcn/81.4.829 10.1079/BJN19910067 10.1073/pnas.0508218103 10.1016/j.tox.2005.12.016 10.1016/j.freeradbiomed.2004.03.015 10.1016/j.mrfmmm.2005.04.021 10.1093/ajcn/48.1.139 10.1074/jbc.M112145200 10.1079/BJN2000227 10.1158/1055-9965.EPI-05-0950 10.1007/s11745-006-1391-3 10.1073/pnas.93.25.14960 10.1093/ajcn/53.1.120 10.1093/ajcn/53.3.748 10.1024/0300-9831.75.3.179 10.1080/10715760320001634852 10.1079/BJN19780073 10.1002/art.1780301012 10.1093/ajcn/37.6.887 10.1093/ajcn/79.6.1060 10.1016/S0006-291X(05)80950-7 10.1258/000456304322880032 10.1016/S0946-672X(01)80002-X 10.1093/ajcn/36.1.24 10.1016/S0946-672X(11)80013-1 10.1006/abio.2001.5333 10.1080/10715760500108513 10.1038/sj.ejcn.1601789 10.1016/S0009-8981(01)00744-6 10.1016/S0140-6736(00)02490-9 10.1042/BJ20040328 10.1093/carcin/bgi267 10.1006/bbrc.1995.1083 10.1385/BTER:73:2:113 10.1093/ajcn/48.2.316 10.1258/0004563054889990 10.1093/aje/kwj097 10.1016/0003-2697(90)90668-Y 10.1079/BJN19930057 10.1038/sj.ejcn.1601787 10.1097/00007890-199703150-00012 10.1016/S0946-672X(04)80033-6 10.1039/b209585h |
| ContentType | Journal Article |
| Copyright | Copyright © The Authors 2007 2008 INIST-CNRS The Authors |
| Copyright_xml | – notice: Copyright © The Authors 2007 – notice: 2008 INIST-CNRS – notice: The Authors |
| CorporateAuthor | Department of Chemistry Pure and Applied Biochemistry Kemiska institutionen Centrum för Molekylär Proteinvetenskap Lunds universitet Naturvetenskapliga fakulteten Faculty of Science Tillämpad biokemi Lund University Center for Molecular Protein Science |
| CorporateAuthor_xml | – name: Naturvetenskapliga fakulteten – name: Department of Chemistry – name: Lund University – name: Tillämpad biokemi – name: Center for Molecular Protein Science – name: Pure and Applied Biochemistry – name: Kemiska institutionen – name: Faculty of Science – name: Lunds universitet – name: Centrum för Molekylär Proteinvetenskap |
| DBID | FBQ BSCLL AAYXX CITATION IQODW CGR CUY CVF ECM EIF NPM 3V. 7QP 7RV 7T5 7X2 7X7 7XB 88E 8C1 8FE 8FH 8FI 8FJ 8FK 8G5 ABUWG AEUYN AFKRA AN0 ATCPS AZQEC BENPR BHPHI CCPQU DWQXO FYUFA GHDGH GNUQQ GUQSH H94 HCIFZ K9. KB0 M0K M0S M1P M2O MBDVC NAPCQ PHGZM PHGZT PJZUB PKEHL PPXIY PQEST PQQKQ PQUKI Q9U M7N 7S9 L.6 7X8 ADTPV AOWAS D95 |
| DOI | 10.1017/S0007114507825153 |
| DatabaseName | AGRIS Istex CrossRef Pascal-Francis Medline MEDLINE MEDLINE (Ovid) MEDLINE MEDLINE PubMed ProQuest Central (Corporate) Calcium & Calcified Tissue Abstracts Nursing & Allied Health Database Immunology Abstracts Agricultural Science Collection Health & Medical Collection ProQuest Central (purchase pre-March 2016) Medical Database (Alumni Edition) Public Health Database ProQuest SciTech Collection ProQuest Natural Science Collection Hospital Premium Collection Hospital Premium Collection (Alumni Edition) ProQuest Central (Alumni) (purchase pre-March 2016) Research Library (Alumni) ProQuest Central (Alumni) ProQuest One Sustainability ProQuest Central UK/Ireland British Nursing Database Agricultural & Environmental Science Collection ProQuest Central Essentials ProQuest Central Natural Science Collection ProQuest One ProQuest Central Proquest Health Research Premium Collection Health Research Premium Collection (Alumni) ProQuest Central Student ProQuest Research Library AIDS and Cancer Research Abstracts SciTech Premium Collection ProQuest Health & Medical Complete (Alumni) Nursing & Allied Health Database (Alumni Edition) Agriculture Science Database ProQuest Health & Medical Collection Medical Database ProQuest Research Library Research Library (Corporate) Nursing & Allied Health Premium ProQuest Central Premium ProQuest One Academic (New) ProQuest Health & Medical Research Collection ProQuest One Academic Middle East (New) ProQuest One Health & Nursing ProQuest One Academic Eastern Edition (DO NOT USE) ProQuest One Academic ProQuest One Academic UKI Edition ProQuest Central Basic Algology Mycology and Protozoology Abstracts (Microbiology C) AGRICOLA AGRICOLA - Academic MEDLINE - Academic SwePub SwePub Articles SWEPUB Lunds universitet |
| DatabaseTitle | CrossRef MEDLINE Medline Complete MEDLINE with Full Text PubMed MEDLINE (Ovid) Agricultural Science Database Research Library Prep ProQuest Central Student ProQuest One Academic Middle East (New) ProQuest Central Essentials ProQuest Health & Medical Complete (Alumni) ProQuest Central (Alumni Edition) SciTech Premium Collection ProQuest One Community College ProQuest One Health & Nursing Research Library (Alumni Edition) ProQuest Natural Science Collection ProQuest Central ProQuest One Sustainability ProQuest Health & Medical Research Collection Health Research Premium Collection Health and Medicine Complete (Alumni Edition) Natural Science Collection ProQuest Central Korea Health & Medical Research Collection Agricultural & Environmental Science Collection AIDS and Cancer Research Abstracts ProQuest Research Library ProQuest Central (New) ProQuest Medical Library (Alumni) ProQuest Public Health ProQuest Central Basic ProQuest One Academic Eastern Edition Agricultural Science Collection British Nursing Index with Full Text ProQuest Nursing & Allied Health Source ProQuest Hospital Collection Health Research Premium Collection (Alumni) ProQuest SciTech Collection ProQuest Hospital Collection (Alumni) Nursing & Allied Health Premium ProQuest Health & Medical Complete ProQuest Medical Library ProQuest One Academic UKI Edition Immunology Abstracts ProQuest Nursing & Allied Health Source (Alumni) ProQuest One Academic Calcium & Calcified Tissue Abstracts ProQuest One Academic (New) ProQuest Central (Alumni) Algology Mycology and Protozoology Abstracts (Microbiology C) AGRICOLA AGRICOLA - Academic MEDLINE - Academic |
| DatabaseTitleList | MEDLINE MEDLINE - Academic Algology Mycology and Protozoology Abstracts (Microbiology C) AGRICOLA Agricultural Science Database CrossRef |
| Database_xml | – sequence: 1 dbid: NPM name: PubMed url: https://proxy.k.utb.cz/login?url=http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=PubMed sourceTypes: Index Database – sequence: 2 dbid: EIF name: MEDLINE url: https://proxy.k.utb.cz/login?url=https://www.webofscience.com/wos/medline/basic-search sourceTypes: Index Database – sequence: 3 dbid: BENPR name: ProQuest Central url: https://www.proquest.com/central sourceTypes: Aggregation Database – sequence: 4 dbid: FBQ name: AGRIS url: http://www.fao.org/agris/Centre.asp?Menu_1ID=DB&Menu_2ID=DB1&Language=EN&Content=http://www.fao.org/agris/search?Language=EN sourceTypes: Publisher |
| DeliveryMethod | fulltext_linktorsrc |
| Discipline | Diet & Clinical Nutrition |
| DocumentTitleAlternate | G. Ravn-Haren et al. Short-term effects of selenium sources |
| EISSN | 1475-2662 |
| EndPage | 892 |
| ExternalDocumentID | oai_portal_research_lu_se_publications_44def329_ed09_43c4_b2ab_8f349fd4617e oai_lup_lub_lu_se_44def329_ed09_43c4_b2ab_8f349fd4617e 1496037701 17888202 20214781 10_1017_S0007114507825153 ark_67375_6GQ_VGZ47ZFJ_C US201300885665 |
| Genre | Randomized Controlled Trial Research Support, Non-U.S. Gov't Journal Article |
| GroupedDBID | --- -E. -~X .FH 09C 09E 0E1 0R~ 23N 2WC 3EH 3V. 4.4 5GY 5RE 5VS 6J9 6PF 74X 74Y 7RV 7X2 7X7 7~V 88E 8C1 8FE 8FH 8FI 8FJ 8G5 8R4 8R5 9M5 AAAZR AABES AABWE AACJH AAEED AAFWJ AAGFV AAIKC AAKTX AAMNQ AAMNW AARAB AASVR AATID AAUIS AAUKB AAWTL AAYJJ AAZAQ ABBJB ABBXD ABFBI ABGDZ ABJNI ABKKG ABKMT ABMWE ABMYL ABQTM ABROB ABUWG ABWCF ABZCX ABZUI ACBEK ACBMC ACCHT ACGFS ACIMK ACPRK ACQFJ ACREK ACUIJ ACUYZ ACWGA ACYZP ACZBM ACZUX ACZWT ADAZD ADBBV ADDNB ADFEC ADFRT ADGEJ ADKIL ADOCW ADVJH AEBAK AEHGV AEMTW AENEX AENGE AEPLO AEYHU AEYYC AFFNX AFFUJ AFKQG AFKRA AFLOS AFLVW AFOSN AFRAH AFRIC AFUTZ AGABE AGJUD AGOOT AHIPN AHLTW AHMBA AHQXX AHRGI AIGNW AIHIV AIOIP AISIE AJ7 AJCYY AJPFC AJQAS ALMA_UNASSIGNED_HOLDINGS ALWZO AN0 ANPSP AQJOH ATCPS ATUCA AUXHV AZGZS AZQEC BAWUL BBLKV BENPR BHPHI BKEYQ BLZWO BNQBC BPHCQ BQFHP BRIRG BVXVI C0O C1A CAG CBIIA CCPQU CCQAD CFAFE CHEAL CJCSC COF CS3 DIK DOHLZ DU5 DWQXO E3Z EBS EJD EX3 F5P FYUFA GNUQQ GUQSH HCIFZ HG- HH5 HMCUK HZ~ I.6 IH6 IOEEP IOO IS6 I~P J36 J38 J3A JHPGK JKPOH JQKCU JVRFK KCGVB KFECR L7B L98 LW7 M-V M0K M1P M2O M7~ NAPCQ NIKVX O9- OK1 OVD P6G PCD PQQKQ PROAC PSQYO Q2X RAMDC RCA RIG ROL RR0 S6- S6U SAAAG SJN SY4 T9M TEORI TR2 UCJ UKHRP UT1 UU6 WFFJZ WH7 WOW WXU WYP Y6R ZA5 ZMEZD ZYDXJ ~KM -1D -1F -2P -2V -~6 -~N .55 .GJ 354 3O- 53G 6~7 AANRG ABBZL ABVFV ACETC ADOVH ADOVT AEBPU AENCP AEQTP AGLWM AI. AKZCZ ALEEW ARABE ARZZG AYIQA BCGOX BESQT BGHMG BJBOZ BMAJL CCUQV CDIZJ CFBFF CGQII EGQIC FBQ HST I.7 I.9 KAFGG KC5 LHUNA M8. NMFBF NZEOI OHT UAP VH1 WQ3 WXY X7M ZCG ZDLDU ZGI ZJOSE ZXP ~V1 AAHBH AATMM ABVKB ABVZP ABXAU ABXHF ACDLN ACRPL ADNMO AEMFK AEUYN AFZFC AGQPQ AKMAY BSCLL PHGZM PHGZT PJZUB PPXIY PUEGO AAKNA AAYXX ABHFL ACEJA ACOZI ANOYL CITATION IQODW CGR CUY CVF ECM EIF NPM 7QP 7T5 7XB 8FK H94 K9. MBDVC PKEHL PQEST PQUKI Q9U M7N 7S9 L.6 7X8 ADTPV AOWAS D95 |
| ID | FETCH-LOGICAL-c726t-d24a7ae888a57ee96656d7504a2d53d5c296a375e3f6b44da648a55f116865f13 |
| IEDL.DBID | 7X7 |
| ISSN | 0007-1145 1475-2662 |
| IngestDate | Thu Aug 21 06:46:31 EDT 2025 Thu Jul 03 05:07:42 EDT 2025 Thu Jul 10 19:02:06 EDT 2025 Tue Aug 05 10:33:48 EDT 2025 Fri Jul 11 15:48:20 EDT 2025 Sat Aug 23 13:01:09 EDT 2025 Mon Jul 21 06:04:57 EDT 2025 Mon Jul 21 09:11:10 EDT 2025 Tue Jul 01 03:49:54 EDT 2025 Thu Apr 24 23:12:04 EDT 2025 Sun Aug 31 06:50:38 EDT 2025 Wed Dec 27 19:05:52 EST 2023 Wed Mar 13 05:47:53 EDT 2024 |
| IsDoiOpenAccess | false |
| IsOpenAccess | true |
| IsPeerReviewed | true |
| IsScholarly | true |
| Issue | 4 |
| Keywords | Glutathione peroxidase Oxidative defence Selenium Blood lipids Electrophile response elements Short term Yeast Enzyme Lipids Response element Blood Vertebrata Regulation(control) Mammalia Peroxidases Selenium: Oxidative defence: Electrophile response elements: Glutathione peroxidase: Blood lipids Oxidoreductases Milk |
| Language | English |
| License | https://www.cambridge.org/core/terms CC BY 4.0 |
| LinkModel | DirectLink |
| MergedId | FETCHMERGED-LOGICAL-c726t-d24a7ae888a57ee96656d7504a2d53d5c296a375e3f6b44da648a55f116865f13 |
| Notes | istex:D668E9F45FE3C76A175291747D0F2496A38C9696 ark:/67375/6GQ-VGZ47ZFJ-C ArticleID:82515 PII:S0007114507825153 Abbreviations: CRP, C-reactive protein; GPX, glutathione peroxidase; GR, glutathione reductase; GSH, glutathione; GST, glutathione S-transferase; SelP, selenoprotein P ObjectType-Article-1 SourceType-Scholarly Journals-1 ObjectType-Feature-2 content type line 14 content type line 23 ObjectType-Undefined-3 |
| OpenAccessLink | https://www.cambridge.org/core/services/aop-cambridge-core/content/view/63C136CF7E99E7472C7AC8A3A9A3F5FE/S0007114507825153a.pdf/div-class-title-a-short-term-intervention-trial-with-selenate-selenium-enriched-yeast-and-selenium-enriched-milk-effects-on-oxidative-defence-regulation-div.pdf |
| PMID | 17888202 |
| PQID | 213829490 |
| PQPubID | 5629 |
| PageCount | 10 |
| ParticipantIDs | swepub_primary_oai_portal_research_lu_se_publications_44def329_ed09_43c4_b2ab_8f349fd4617e swepub_primary_oai_lup_lub_lu_se_44def329_ed09_43c4_b2ab_8f349fd4617e proquest_miscellaneous_70370522 proquest_miscellaneous_47700807 proquest_miscellaneous_19924828 proquest_journals_213829490 pubmed_primary_17888202 pascalfrancis_primary_20214781 crossref_primary_10_1017_S0007114507825153 crossref_citationtrail_10_1017_S0007114507825153 istex_primary_ark_67375_6GQ_VGZ47ZFJ_C fao_agris_US201300885665 cambridge_journals_10_1017_S0007114507825153 |
| ProviderPackageCode | CITATION AAYXX |
| PublicationCentury | 2000 |
| PublicationDate | 2008-04-01 |
| PublicationDateYYYYMMDD | 2008-04-01 |
| PublicationDate_xml | – month: 04 year: 2008 text: 2008-04-01 day: 01 |
| PublicationDecade | 2000 |
| PublicationPlace | Cambridge, UK |
| PublicationPlace_xml | – name: Cambridge, UK – name: Wallingford – name: England – name: Cambridge |
| PublicationTitle | British journal of nutrition |
| PublicationTitleAlternate | Br J Nutr |
| PublicationYear | 2008 |
| Publisher | Cambridge University Press |
| Publisher_xml | – name: Cambridge University Press |
| References | S0007114507825153_ref51 S0007114507825153_ref52 S0007114507825153_ref50 S0007114507825153_ref15 S0007114507825153_ref16 S0007114507825153_ref13 S0007114507825153_ref14 S0007114507825153_ref11 S0007114507825153_ref12 S0007114507825153_ref10 S0007114507825153_ref19 S0007114507825153_ref17 Lyhne (S0007114507825153_ref21) S0007114507825153_ref18 Ip (S0007114507825153_ref2) 1986; 206 S0007114507825153_ref41 S0007114507825153_ref48 S0007114507825153_ref49 S0007114507825153_ref46 Andersen (S0007114507825153_ref30) 1996; 4 S0007114507825153_ref47 S0007114507825153_ref44 S0007114507825153_ref45 S0007114507825153_ref42 S0007114507825153_ref43 Ratnasinghe (S0007114507825153_ref7) 2000; 60 S0007114507825153_ref1 S0007114507825153_ref9 S0007114507825153_ref6 S0007114507825153_ref5 S0007114507825153_ref4 S0007114507825153_ref3 S0007114507825153_ref37 S0007114507825153_ref38 S0007114507825153_ref35 S0007114507825153_ref36 S0007114507825153_ref33 S0007114507825153_ref34 S0007114507825153_ref31 S0007114507825153_ref39 Pruszkowski (S0007114507825153_ref32) 2004 S0007114507825153_ref26 S0007114507825153_ref27 S0007114507825153_ref24 S0007114507825153_ref25 S0007114507825153_ref22 S0007114507825153_ref23 S0007114507825153_ref20 Hu (S0007114507825153_ref8) 2003; 63 Mutanen (S0007114507825153_ref40) 1986; 56 S0007114507825153_ref28 S0007114507825153_ref29 |
| References_xml | – ident: S0007114507825153_ref46 doi: 10.1093/ajcn/81.4.829 – ident: S0007114507825153_ref19 doi: 10.1079/BJN19910067 – ident: S0007114507825153_ref5 doi: 10.1073/pnas.0508218103 – ident: S0007114507825153_ref47 doi: 10.1016/j.tox.2005.12.016 – ident: S0007114507825153_ref26 doi: 10.1016/j.freeradbiomed.2004.03.015 – ident: S0007114507825153_ref23 doi: 10.1016/j.mrfmmm.2005.04.021 – ident: S0007114507825153_ref16 doi: 10.1093/ajcn/48.1.139 – ident: S0007114507825153_ref28 doi: 10.1074/jbc.M112145200 – ident: S0007114507825153_ref45 doi: 10.1079/BJN2000227 – ident: S0007114507825153_ref38 doi: 10.1158/1055-9965.EPI-05-0950 – ident: S0007114507825153_ref29 doi: 10.1007/s11745-006-1391-3 – ident: S0007114507825153_ref27 doi: 10.1073/pnas.93.25.14960 – ident: S0007114507825153_ref14 doi: 10.1093/ajcn/53.1.120 – ident: S0007114507825153_ref17 doi: 10.1093/ajcn/53.3.748 – ident: S0007114507825153_ref39 doi: 10.1024/0300-9831.75.3.179 – ident: S0007114507825153_ref4 doi: 10.1080/10715760320001634852 – ident: S0007114507825153_ref10 doi: 10.1079/BJN19780073 – ident: S0007114507825153_ref43 doi: 10.1002/art.1780301012 – ident: S0007114507825153_ref12 doi: 10.1093/ajcn/37.6.887 – ident: S0007114507825153_ref35 doi: 10.1093/ajcn/79.6.1060 – volume-title: Danskernes Kostvaner 2000-2002. Hovedresultater ident: S0007114507825153_ref21 – ident: S0007114507825153_ref24 doi: 10.1016/S0006-291X(05)80950-7 – ident: S0007114507825153_ref51 doi: 10.1258/000456304322880032 – volume-title: Selenium Determination in Blood and Serum with ICP-MS year: 2004 ident: S0007114507825153_ref32 – ident: S0007114507825153_ref41 doi: 10.1016/S0946-672X(01)80002-X – ident: S0007114507825153_ref11 doi: 10.1093/ajcn/36.1.24 – ident: S0007114507825153_ref15 doi: 10.1016/S0946-672X(11)80013-1 – ident: S0007114507825153_ref33 doi: 10.1006/abio.2001.5333 – ident: S0007114507825153_ref48 doi: 10.1080/10715760500108513 – volume: 60 start-page: 6381 year: 2000 ident: S0007114507825153_ref7 article-title: Glutathione peroxidase codon 198 polymorphism variant increases lung cancer risk publication-title: Cancer Res – ident: S0007114507825153_ref22 doi: 10.1038/sj.ejcn.1601789 – ident: S0007114507825153_ref50 doi: 10.1016/S0009-8981(01)00744-6 – volume: 63 start-page: 3347 year: 2003 ident: S0007114507825153_ref8 article-title: Role of glutathione peroxidase 1 in breast cancer: loss of heterozygosity and allelic differences in the response to selenium publication-title: Cancer Res – ident: S0007114507825153_ref1 doi: 10.1016/S0140-6736(00)02490-9 – ident: S0007114507825153_ref6 doi: 10.1042/BJ20040328 – ident: S0007114507825153_ref9 doi: 10.1093/carcin/bgi267 – volume: 4 start-page: S129 year: 1996 ident: S0007114507825153_ref30 article-title: Typiske vægte for madvarer (Typical weights for foods) publication-title: Scand J Nutr – ident: S0007114507825153_ref37 – ident: S0007114507825153_ref25 doi: 10.1006/bbrc.1995.1083 – volume: 206 start-page: 431 year: 1986 ident: S0007114507825153_ref2 article-title: The chemopreventive role of selenium in carcinogenesis publication-title: Adv Exp Med Biol – ident: S0007114507825153_ref44 doi: 10.1385/BTER:73:2:113 – ident: S0007114507825153_ref18 doi: 10.1093/ajcn/48.2.316 – ident: S0007114507825153_ref52 doi: 10.1258/0004563054889990 – ident: S0007114507825153_ref49 doi: 10.1093/aje/kwj097 – ident: S0007114507825153_ref34 doi: 10.1016/0003-2697(90)90668-Y – ident: S0007114507825153_ref36 – ident: S0007114507825153_ref13 doi: 10.1079/BJN19930057 – volume: 56 start-page: 297 year: 1986 ident: S0007114507825153_ref40 article-title: Bioavailability of selenium in mushrooms, Boletus edulis, to young women publication-title: Int J Vitam Nutr Res – ident: S0007114507825153_ref20 doi: 10.1038/sj.ejcn.1601787 – ident: S0007114507825153_ref3 doi: 10.1097/00007890-199703150-00012 – ident: S0007114507825153_ref42 doi: 10.1016/S0946-672X(04)80033-6 – ident: S0007114507825153_ref31 doi: 10.1039/b209585h |
| SSID | ssj0008109 |
| Score | 2.1557105 |
| Snippet | Increased Se intakes have been associated with decreased risk of cancer and CVD. Several mechanisms have been proposed, including antioxidant effects through... |
| SourceID | swepub proquest pubmed pascalfrancis crossref istex fao cambridge |
| SourceType | Open Access Repository Aggregation Database Index Database Enrichment Source Publisher |
| StartPage | 883 |
| SubjectTerms | administration & dosage Adolescent Adult adults analysis Animals antioxidant activity Antioxidants Antioxidants - administration & dosage Antioxidants - analysis Base Sequence Bioavailability Biological and medical sciences Biomarkers Biomarkers - blood Blood blood chemistry Blood lipids blood platelets C-Reactive Protein C-Reactive Protein - analysis Cancer Cholesterol Cholesterol - blood Cholesterol, HDL Cholesterol, HDL - blood Cholesterol, LDL Cholesterol, LDL - blood Colleges & universities Cross-Over Studies Dairy products Detoxification Diet dietary mineral supplements dietary minerals Dietary Supplements DNA Primers DNA Primers - genetics Double-Blind Method drug effects Electrophile response elements enriched foods Erythrocytes Feeding. Feeding behavior Food, Fortified Fundamental and applied biological sciences. Psychology Gene expression Gene Expression - drug effects genetics Glutathione peroxidase glutathione reductase (NADPH) glutathione transferase glutathione-disulfide reductase Health risks Health Sciences high density lipoprotein Humans Hälsovetenskap leukocytes Leukocytes - metabolism lipid peroxidation Lipids Lipids - blood low density lipoprotein Male Medical and Health Sciences Medicin och hälsovetenskap men metabolism Milk Molecular Sequence Data Nutrition and Dietetics Nutrition research nutritional intervention Näringslära Näringslära och dietkunskap oxidation Oxidation-Reduction Oxidative defence Plasma protein synthesis Reverse Transcriptase Polymerase Chain Reaction Risk factors Risk reduction selenates Selenium Selenium - administration & dosage Selenium - blood Studies thrombocytes Vertebrates: anatomy and physiology, studies on body, several organs or systems Yeast, Dried Yeasts |
| Title | A short-term intervention trial with selenate, selenium-enriched yeast and selenium-enriched milk: effects on oxidative defence regulation |
| URI | https://www.cambridge.org/core/product/identifier/S0007114507825153/type/journal_article https://api.istex.fr/ark:/67375/6GQ-VGZ47ZFJ-C/fulltext.pdf https://www.ncbi.nlm.nih.gov/pubmed/17888202 https://www.proquest.com/docview/213829490 https://www.proquest.com/docview/19924828 https://www.proquest.com/docview/47700807 https://www.proquest.com/docview/70370522 https://lup.lub.lu.se/record/1206490 oai:portal.research.lu.se:publications/44def329-ed09-43c4-b2ab-8f349fd4617e |
| Volume | 99 |
| hasFullText | 1 |
| inHoldings | 1 |
| isFullTextHit | |
| isPrint | |
| link | http://utb.summon.serialssolutions.com/2.0.0/link/0/eLvHCXMwjV1bb9MwFLbY9sIL4r4wKH5Ae0CLaB07TnhBo7Sb9lBxK6r6YjmxvVXr0tKLNP4Cv5pzEjfroKt4SFvF9lFjn5yL_ek7hLxpJeAEMwtJTuKSkFtIWLXITMg5Y7lzNhGmZPvsxad9fjYQA4_NmXtY5comlobaTHLcI3_HkCwv5Wnzw_RniEWj8HDVV9DYIXvIXIZKLQd1vgXOziM8kAexxcXqULNkjEbfCvfQQ4JLj9apFW65qB2nJxC44pxfI3BSz2HuXFX0YlNU-hflaOmmug_JAx9f0uNKIR6Re7Z4TIJPI7ugh9STgI5pb8XB_4T8PqbzC4jBQ7TRdLQGgaRlRQ-KO7UUazIVEJYeVb9Gy6sQFA9hpIb-wvI_VBdmQ9vVaHz5nnrQCAWZk-uRKbnGqbEOrQqd2XNfQuwp6Xc739unoS_QEOaSxYvQMK6ltpBEayGthcxJxAb54jUzIjIiZ2msIyls5OKMc6NjDj2Fa7XiJIav6BnZLSaF3SeUcceNiVMnIsd5HmvIPA3Pc5YZFuWZDshRvT7Kv2ZzVUHUpPpnOQPSXC2hyj3ZOdbcGG8b8rYeMq2YPrZ13ge9UPocLLHqf2N4_gv2GmJjEZDDUllqIXp2ieg5KVR88kX9OBlyOeyeqXZAGre0qR7AkMdOJq2AHKzU6-aR6_cgIK_rVjAJeM6jCztZwqykkFRDJn13Dy4lpgry7h7gCGQTYvOAPK_0-mZOcNME_mJAOpWi1y3IVD5eTuHK4FJzq2DNrYtYqqxppopHOVcZ05lKXMRTZziEzzYgww1yquRTecarCy9vuraV_V_CX2ydwgNyv4L_IBDrJdldzJb2FcSYi6xRWhL4TNqtBtn72Ol9_voHzvx5HQ |
| linkProvider | ProQuest |
| linkToHtml | http://utb.summon.serialssolutions.com/2.0.0/link/0/eLvHCXMwtV1LbxMxELbacoAL4t2l0PoAPaCuyHrtfSAhVKVN0weREA2qcjHetd1GTZOQh2j_Av-F_8jMvppCGnHpYbWr2B5tPON52LPfEPLGi8AIJgaCnMhGLjcQsCqRaJdzxlJrTSR0hvbZCpptfnAiTpbI7_JbGEyrLHVipqj1IMU98vcMwfJiHtc-DX-4WDQKD1fLChq5VByaq58QsY0_7u8Ae98y1tg9rjfdoqiAm4YsmLiacRUqA4GfEqEx4O2LQCPGuWJa-FqkLA6UHwrj2yDhXKuAQ09hPS-IArj5QHeZ3OM-rEz8ML1-nVESeUVGCeIuelyUh6gZQjXacvgNLTK4EP4slMMNk7hs1QAcZeTxJSZqqjHwyuZFNuZ5wX9BnGZmsfGIPCz8WbqdC-BjsmT6T4iz0zUTukkL0NEebZWY_0_Jr206PgOf30WbQLszKZc0qyBCcWeYYg2oPrjBW_lTd3rhgqBj2qqmV1huiKq-ntN20e2df6BFkgoFmoPLrs6wzak2FrUYHZnTomTZM9K-E949Jyv9Qd-sEsq45VoHsRW-5TwNFES6mqcpSzTz00Q5ZKvijyyW9VjmKXGh_IedDqmVLJRpAa6ONT56i4a8q4YMc2SRRZ1XQS6kOgXNL9tfGZ43g30AX1w4ZDMTloqIGp1jtl4oZLD3RX7b6_Cw0ziQdYes35CmagBD3Lww8hyyVorX9V-u1p1DNqpWUEF4rqT6ZjCFWYkhiIfI_fYePAwxNAlv7wGGJ6xBLOCQF7lcX88JbtLAKzpkNxf0qgWR0XvTIVwJXHJsJPDcWJ_F0uhaLLmfcpkwlcjI-jy2moO7bhzSmUMnD3ZlgbB1VtAbzmyd_xfxlwuncIPcbx5_PpJH-63DNfIgTz3CJLBXZGUymprX4N9OkvVMq1Dy_a7V2B-fZ7JD |
| linkToPdf | http://utb.summon.serialssolutions.com/2.0.0/link/0/eLvHCXMwtV1JbxMxFLbaIiEuiL1DofUBekAdNfHY4xkkhKqk6YYqEARVubiesd1GTZOQRbR_gX_Ev-O92dJCG3HpYZQoXpT4Pb_F_vI9Qt7UI3CCiYUkJ3KRzy0krFokxuecsdQ5GwmTsX0ehrttvn8kjhbI7_K_MAirLG1iZqjNIMUz8k2GZHkxj2ubrkBFfG62Pg5_-FhACi9ay2oauYYc2MufkL2NP-w1QdRvGWttf2vs-kWBAT-VLJz4hnEttYUkUAtpLUT-IjTId66ZEYERKYtDHUhhAxcmnBsdcugpXL0eRiG8BDDvIrknAxnhFosaM3RJVC_QJcjBWOeivFDN2KrRr8Nn6J0hnAiu0jpcc4-LTg8gaEZ5XyBoU49Bbi4vuHFTRPwX3WnmIluPyMMitqVbuTI-Jgu2_4R4za6d0HVaEJD26GHJ__-U_Nqi41OI_330D7R7BX5Js2oiFE-JKdaD6kNIvJG_607PfVB6hLAaeomlh6jumxvazru9s_e0AKxQmHNw0TUZzzk11qFFoyN7UpQve0badyK752SpP-jbZUIZd9yYMHYicJynoYas1_A0ZYlhQZpoj2xU8lHFFh-rHB4n1T_i9EitFKFKC6J1rPfRmzfkXTVkmLOMzOu8DHqh9Al4AdX-yvDuGXwFxOXCI-uZslST6NEZIvekUOHOF_V9p8Nlp7WvGh5ZvaZN1QCGHHoyqntkpVSv2U-u9qBH1qpWMEd4x6T7djCFVYkhoYcs_vYeXEpMU-TtPcAJyRrkBR55kev1bE3wwAa-oke2c0WvWpAlvTcdwpPAo8ZWgcytC1isrKnFigcpVwnTiYpcwGNnOITu1iOdG-bJE19VsG2dFvMNrxyj_9fkL-cu4Rq5DwZMfdo7PFghD3IUEuLBXpGlyWhqX0OoO0lWM6NCyfFdW7E_b9C2eQ |
| openUrl | ctx_ver=Z39.88-2004&ctx_enc=info%3Aofi%2Fenc%3AUTF-8&rfr_id=info%3Asid%2Fsummon.serialssolutions.com&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=A+short-term+intervention+trial+with+selenate%2C+selenium-enriched+yeast+and+selenium-enriched+milk%3A+effects+on+oxidative+defence+regulation&rft.jtitle=British+journal+of+nutrition&rft.au=Ravn-Haren%2C+Gitte&rft.au=B%C3%BCgel%2C+Susanne&rft.au=Krath%2C+Britta+N&rft.au=Hoac%2C+Tien&rft.date=2008-04-01&rft.pub=Cambridge+University+Press&rft.issn=0007-1145&rft.eissn=1475-2662&rft.volume=99&rft.issue=4&rft.spage=883&rft_id=info:doi/10.1017%2FS0007114507825153&rft.externalDBID=HAS_PDF_LINK&rft.externalDocID=1496037701 |
| thumbnail_l | http://covers-cdn.summon.serialssolutions.com/index.aspx?isbn=/lc.gif&issn=0007-1145&client=summon |
| thumbnail_m | http://covers-cdn.summon.serialssolutions.com/index.aspx?isbn=/mc.gif&issn=0007-1145&client=summon |
| thumbnail_s | http://covers-cdn.summon.serialssolutions.com/index.aspx?isbn=/sc.gif&issn=0007-1145&client=summon |