CLEC9A modulates macrophage-mediated neutrophil recruitment in response to heat-killed Mycobacterium tuberculosis H37Ra - Cheng, An-Chieh; Yang, Kuang-Yao; Chen, Nien-Jung; Hsu, Tsui-Ling; Jou, Ruwen; Hsieh, Shie-Liang; Tseng, Ping-Hui (PloS one, 2017)
Keywords: Adaptive immunity; Animals; Antigens; Bacteria; Biochemistry; Biology and Life Sciences; Cell activation; Cell Line; Cell walls; Chemokines; Cytokines; Development and progression; Fc receptors; Fusion protein; Gene Knockdown Techniques; Genetic aspects; Genomics; Granulocytes; Hot Temperature; Humans; IL-1β; Immune response; Immune system; Immunity; Immunology; Infections; Infectious diseases; Infiltration; Innate immunity; Kinases; Lectins; Lectins, C-Type - genetics; Lectins, C-Type - physiology; Leukemia; Life sciences; Lungs; Macrophages; Macrophages - enzymology; Macrophages - physiology; Male; MAP kinase; Medicine and Health Sciences; Mice; Mice, Inbred C57BL; Molecular biology; Mycobacterium tuberculosis; Mycobacterium tuberculosis - physiology; Neutrophils; Neutrophils - cytology; Pathogenesis; Pathogenicity; Pathogens; Pattern recognition; Pattern recognition receptors; Physiological aspects; Polysaccharides; Protein Kinases - metabolism; Proteins; Receptors; Receptors, Mitogen - genetics; Receptors, Mitogen - physiology; Research and Analysis Methods; Signal Transduction; Signaling; Syk protein; Tuberculosis; Taiwan