Aged blood factors decrease cellular responses associated with delayed gingival wound repair - Saldías, María Paz; Fernández, Christian; Morgan, Alejandra; Díaz, Catalina; Morales, Diego; Jaña, Fabián; Gómez, Alvaro; Silva, Alonso; Briceño, Fernanda; Oyarzún, Alejandro; Maldonado, Felipe; Cerda, Oscar; et al. (PloS one, 2017)
Keywords: Actin; Age; Age Factors; Aged; Aged, 80 and over; Aging; Aging - blood; Analysis; Animals; beta-Galactosidase - metabolism; Biological activity; Biology; Biology and Life Sciences; Blood; Blood platelets; Cell adhesion & migration; Cell Cycle - drug effects; Cell Differentiation - drug effects; Cell Proliferation; Connective tissues; Cytokines; Cytokines - blood; Cytokines - pharmacology; Cytometry; Dentistry; Deoxyribonucleic acid; Diabetes; Differentiation; Disease Models, Animal; DNA; Extracellular matrix; Fibroblasts; Fibroblasts - metabolism; Flow cytometry; Galactosidase; Genetic aspects; Gingiva; Gingiva - drug effects; Gingiva - metabolism; Gingiva - pathology; Growth factors; Histone H2A; Humans; Immunofluorescence; In vitro methods and tests; Inflammation; Inflammation Mediators - blood; Inflammation Mediators - pharmacology; Interleukin 6; Male; Medicine; Medicine and Health Sciences; Middle Aged; Monocyte chemoattractant protein 1; Myofibroblasts - cytology; Myofibroblasts - drug effects; Myofibroblasts - metabolism; Physiological aspects; Platelet-derived growth factor; Rats; Repair; Senescence; Serum; Smooth muscle; Staining; Tissues; Tumor necrosis factor; Tumor necrosis factor-TNF; Vascular endothelial growth factor; Wound healing; Wound Healing - drug effects; β-Galactosidase; Chile