Epstein–Barr virus-induced gene 3 commits human mesenchymal stem cells to differentiate into chondrocytes via endoplasmic reticulum stress sensor - Zhang, Tong; Yamagata, Kaoru; Iwata, Shigeru; Sonomoto, Koshiro; Trimova, Gulzhan; Nguyen, Anh Phuong; Hao, He; Shan, Yu; Nguyen, Mai-Phuong; Nakayamada, Shingo; Tanaka, Yoshiya (PloS one, 2022)
Keywords: Accumulation; Analysis; Antibodies; Arthritis; Biology and Life Sciences; Bone marrow; Cartilage; Cell cycle; Cell differentiation; Cell Differentiation - genetics; Chondrocytes; Chondrocytes - cytology; Chondrogenesis; Chondrogenesis - genetics; Control; Cytokines; Differentiation; EBI3 protein; Endoplasmic reticulum; Endoplasmic Reticulum Stress - genetics; Endoribonucleases - genetics; Endoribonucleases - metabolism; Epstein-Barr virus; Health aspects; Humans; Identification and classification; IL-1β; Inositol; Inositols; Interleukin 27; Interleukins; Interleukins - genetics; Interleukins - physiology; Kinases; Medicine and Health Sciences; Mesenchymal stem cells; Mesenchymal Stem Cells - cytology; Minor Histocompatibility Antigens - genetics; Minor Histocompatibility Antigens - physiology; Phenylbutyric acid; Prevention; Protein Serine-Threonine Kinases - genetics; Protein Serine-Threonine Kinases - metabolism; Proteins; Research and Analysis Methods; Rheumatoid arthritis; Risk factors; Sensors; Stem cells; Stress; Stress (Physiology); Tumor necrosis factor-α; Viruses; Japan; United States--US; Chicago Illinois