Pro-inflammatory miR-223 mediates the cross-talk between the IL23 pathway and the intestinal barrier in inflammatory bowel disease - Wang, Huiling; Chao, Kang; Ng, Siew Chien; Bai, Alfa Hc; Yu, Qiao; Yu, Jun; Li, Manying; Cui, Yi; Chen, Minhu; Hu, Ji-Fan; Zhang, Shenghong (Genome Biology, 2016)
Keywords: Animals; Antagomirs; antibodies; Caco-2 Cells; Cell adhesion & migration; Cell Line; Claudins - genetics; Claudins - metabolism; Colitis; Colon; Cytokines; Disease Models, Animal; Gene expression; Gene Expression Profiling - methods; Gene Expression Regulation; Helper cells; Humans; Hypoxia; immunohistochemistry; Inflammation; Inflammatory bowel disease; Inflammatory bowel diseases; Inflammatory Bowel Diseases - chemically induced; Inflammatory Bowel Diseases - drug therapy; Inflammatory Bowel Diseases - genetics; Inflammatory Bowel Diseases - metabolism; Interleukin 23; Interleukin-23 - administration & dosage; Interleukin-23 - genetics; Interleukin-23 - pharmacology; Intestine; Lymphocytes T; Mice; microRNA; MicroRNAs; MicroRNAs - genetics; MicroRNAs - metabolism; Mucosa; multigene family; Oligonucleotide Array Sequence Analysis - methods; Oligonucleotides - administration & dosage; Oligonucleotides - therapeutic use; Pathogenesis; patients; Permeability; Ribonucleic acid; RNA; Rodents; Signal Transduction; staining; sulfonic acids; Tight junctions; Trinitrobenzenesulfonic Acid - adverse effects; Tumor necrosis factor-TNF; Asia; Interleukin 23; miRNA; Pathway; Crohn’s disease; Ulcerative colitis