Kaposi’s sarcoma-associated herpesvirus ORF57 protein protects viral transcripts from specific nuclear RNA decay pathways by preventing hMTR4 recruitment - Ruiz, Julio C.; Hunter, Olga V.; Conrad, Nicholas K. (PLoS pathogens, 2019)
Keywords: B cells; Biological evolution; Biology and life sciences; Biosynthesis; Cell cycle; Cell Line; Cell Nucleus; Deactivation; Decay; Degradation; Exports; Gene expression; Gene Expression Regulation, Viral - genetics; Genes; Genes, Viral - genetics; Genetic engineering; Genomes; GTPase-Activating Proteins - metabolism; GTPase-Activating Proteins - physiology; Health aspects; HEK293 Cells; Herpesvirus 8, Human - metabolism; Herpesvirus 8, Human - pathogenicity; Humans; Inactivation; Kaposis sarcoma; Medicine and Health Sciences; Nuclear Proteins; Pathways; Poly(A)-Binding Protein I - metabolism; Poly(A)-Binding Protein I - physiology; Polyadenine; Protein Binding; Proteins; Quality control; Recruitment; Research and analysis methods; Restoration; Ribonucleic acid; RNA; RNA Helicases - metabolism; RNA Helicases - physiology; RNA interference; RNA polymerase; RNA Stability - genetics; RNA Stability - physiology; RNA viruses; RNA, Nuclear - physiology; RNA, Viral; RNA-Binding Proteins; RNA-mediated interference; RNA-protein interactions; Sarcoma; Tetracyclines; Transcription; Transcription (Genetics); Transcription Factors; Viral Proteins - genetics; Viral Proteins - metabolism; Viral Regulatory and Accessory Proteins - metabolism; Viral Regulatory and Accessory Proteins - physiology; Virus Replication; Viruses; United States--US; Texas