A new highly penetrant form of obesity due to deletions on chromosome 16p11.2 - Walters, R. G.; Jacquemont, S.; Valsesia, A.; de Smith, A. J.; Martinet, D.; Andersson, J.; Falchi, M.; Chen, F.; Andrieux, J.; Lobbens, S.; Delobel, B.; Stutzmann, F.; et al. (Nature (London), 2010)
Keywords: 631/208/2489/144; 631/208/737; 692/699/2743/393; Adolescent; Adult; Age of Onset; Aging; Autism; Biological and medical sciences; Body Mass Index; Case-Control Studies; Child; Chromosome Deletion; Chromosomes; Chromosomes, Human, Pair 16 - genetics; Cognition Disorders; Cognition Disorders - complications; Cognition Disorders - genetics; Cohort Studies; complications; Deletion; Europe; Female; Genetics; Genome-Wide Association Study; Genotype & phenotype; Heterozygote; Human; Humanities and Social Sciences; Humans; Inheritance Patterns; Inheritance Patterns - genetics; letter; Life Sciences; Loci; Male; Medical Genetics and Genomics; Medical sciences; Medicinsk genetik och genomik; Metabolic diseases; multidisciplinary; Mutation; Mutation - genetics; Nuclear power generation; Nucleotides; Obesity; Obesity - complications; Obesity - genetics; Obesity - physiopathology; Pair 16; Penetrance; Penetrants; physiopathology; Public health; Reproducibility of Results; Science; Science (multidisciplinary); Sex Characteristics; Studies; Young Adult; Europe; Deletion; Obesity; Chromosome; Nutritional status; Nutrition disorder