Association of a functional variant downstream of TNFAIP3 with systemic lupus erythematosus - Adrianto, Indra; Wen, Feng; Templeton, Amanda; Wiley, Graham; King, Jarrod B; Lessard, Christopher J; Bates, Jared S; Hu, Yanqing; Kelly, Jennifer A; Kaufman, Kenneth M; Guthridge, Joel M; Alarcón-Riquelme, Marta E; et al. (Nature genetics, 2011)
Keywords: 631/208/205; 631/208/2489/144; 692/699/249/1313/1613; Agriculture; Alliances; Animal Genetics and Genomics; Arthritis; Autoimmune diseases; Base Sequence; Biological and medical sciences; Biomedical and Life Sciences; Biomedicine; Cancer Research; Chromosomes; Disease susceptibility; DNA-Binding Proteins; Female; Fundamental and applied biological sciences. Psychology; Gene Function; Genetic aspects; Genetics; Genetics of eukaryotes. Biological and molecular evolution; Haplotypes; Health aspects; Health sciences; Human Genetics; Humans; Intracellular Signaling Peptides and Proteins - genetics; letter; Linkage Disequilibrium; Lupus; Lupus Erythematosus, Systemic - genetics; Male; Medical sciences; MEDICIN; MEDICINE; Molecular Sequence Data; Nuclear Proteins - genetics; Polymorphism, Single Nucleotide; Quality control; Sarcoidosis. Granulomatous diseases of unproved etiology. Connective tissue diseases. Elastic tissue diseases. Vasculitis; Science; Systemic lupus erythematosus; Tumor necrosis factor; Tumor Necrosis Factor alpha-Induced Protein 3; United Kingdom; Variant; Immunopathology; Connective tissue disease; Skin disease; Association; Systemic lupus erythematosus; Systemic disease; Autoimmune disease