The role of TWEAK/Fn14 signaling in the MPTP-model of Parkinson’s disease - Mustafa, S.; Martin, H.L.; Burkly, L.; Costa, A.; Martins, M.L.; Schwaninger, M.; Teismann, P. (Neuroscience, 2016)
Keywords: Aged; Animals; Blotting, Western; Chromatography, High Pressure Liquid; Cytokine TWEAK; Disease Models, Animal; Female; Fn14; Humans; Immunohistochemistry; Male; Mice; Mice, Inbred C57BL; Mice, Knockout; MPTP; MPTP Poisoning; Neurology; Parkinsonian Disorders - metabolism; Parkinson’s disease; Receptors, Tumor Necrosis Factor - metabolism; Signal Transduction - physiology; Tumor Necrosis Factors - metabolism; tumor-necrosis-factor-alpha; TWEAK; TWEAK Receptor; PBS; COX-2; DOPAC; TBS; TWEAK; tumor-necrosis-factor-alpha; HRP; SNpc; FN14; PCA; TNFR1; NF-κB; PD; TH; TNF-α; SEM; MPTP; HPLC; Parkinson’s disease; phosphate-buffered saline; TNF receptor 1; tumor necrosis factor like weak inducer of apoptosis; tumor necrosis factor-alpha; 3,4-dihydrophenylacetic acid; Tris-buffered saline; horseradish peroxidase; high-performance liquid chromatography; fibroblast growth factor-inducible 14; cycoloxygenase-2; tyrosine hydroxylase; substantia nigra pars compacta; 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine; perchloric acid; nuclear factor-kappaB; standard error of the mean; Fn14