Abnormal structure-function relationships in hereditary dystonia - Carbon, M.; Eidelberg, D. (Neuroscience, 2009)
Keywords: Animals; brain activation; Deep Brain Stimulation; Dystonic Disorders - genetics; Dystonic Disorders - pathology; Dystonic Disorders - physiopathology; Dystonic Disorders - therapy; DYT1; DYT6; Humans; Molecular Chaperones - genetics; motor sequence learning; Neural Pathways - pathology; Neural Pathways - physiopathology; Neurology; positron emission tomography; primary torsion dystonia; Receptors, Dopamine D2 - genetics; Receptors, Dopamine D2 - metabolism; GPi; PTD; VSEQ; motor sequence learning; OT; primary torsion dystonia; SMA; MT; PMC; FDG; brain activation; TDRP; DYT1; DBS; ANOVA; RAC; MSEQ; DYT6; DTI; DYT-RP; positron emission tomography; PET; [ 11C]-raclopride; premotor cortex; onset time; internal globus pallidus; analysis of variance; [ 18F]-fluorodeoxyglucose; movement time; visual sequence learning task; dystonia manifestation-related pattern; torsion dystonia-related pattern; motor sequence learning task; deep brain stimulation; supplementary motor area; diffusion tensor MRI