NUSAP1 promotes pancreatic ductal adenocarcinoma progression by drives the epithelial-mesenchymal transition and reduces AMPK phosphorylation - Liu, Yuan; Tang, Rong; Meng, Qing-Cai; Shi, Si; Xu, Jin; Yu, Xian-Jun; Zhang, Bo; Wang, Wei (BMC cancer, 2024)
Keywords: Adenocarcinoma; Analysis; B cells; Beta cells; Bioinformatics; Biotechnology; Care and treatment; CD8 antigen; Cell cycle; Cell division; Cell migration; Cell proliferation; Chromosomes; Datasets; Development and progression; Differentially expressed genes; DNA microarrays; Functional enrichment analysis; Gene expression; Genes; Genetic aspects; Genomes; Genomics; Health aspects; Heterozygosity; Homologous recombination; Loss of heterozygosity; Lymphocytes T; Medical prognosis; Molecular modelling; Mutation; Nucleoli; Pancreas; Pancreatic cancer; Pancreatic ductal adenocarcinoma; Pathogenesis; Phosphorylation; Prognosis; Protein expression; Protein-protein interactions; Proteins; Protein‒protein interaction; Scientific equipment and supplies industry; Software; Stem cells; Survival analysis; T cells; Transcriptomes; Tumorigenesis; China; United States; Survival analysis; Differentially expressed genes; Functional enrichment analysis; Pancreatic ductal adenocarcinoma; Protein‒protein interaction