MICA polymorphisms and decreased expression of the MICA receptor NKG2D contribute to idiopathic pulmonary fibrosis susceptibility - Aquino-Galvez, Arnoldo; Pérez-Rodríguez, Martha; Camarena, Ángel; Falfan-Valencia, Ramces; Ruiz, Víctor; Montaño, Martha; Barrera, Lourdes; Sada-Ovalle, Isabel; Ramírez, Remedios; Granados, Julio; Pardo, Annie; Selman, Moisés (Human genetics, 2009)
Keywords: Adult; Aged; Analysis; Biological and medical sciences; Biomedical and Life Sciences; Biomedicine; Biopsy; Case-Control Studies; Cell receptors; Cell structures and functions; Cells, Cultured; Classical genetics, quantitative genetics, hybrids; Development and progression; Enzyme-linked immunosorbent assay; Female; Fundamental and applied biological sciences. Psychology; Gene Expression; Gene Frequency; Gene Function; Genetic aspects; Genetic polymorphisms; Genetic Predisposition to Disease; Genetics of eukaryotes. Biological and molecular evolution; Genotype; Histocompatibility Antigens Class I - blood; Histocompatibility Antigens Class I - genetics; HLA-B Antigens - genetics; Human; Human Genetics; Humans; Idiopathic Pulmonary Fibrosis - genetics; Killer cells; Killer Cells, Natural - metabolism; Lung - metabolism; Lung diseases; Lungs; Lymphocytes; Male; Medical sciences; Metabolic Diseases; Middle Aged; Miscellaneous; Molecular and cellular biology; Molecular Medicine; NK Cell Lectin-Like Receptor Subfamily K - genetics; NK Cell Lectin-Like Receptor Subfamily K - metabolism; Original Investigation; Pneumology; Polymorphism, Genetic; Proteins; Pulmonary fibrosis; Respiratory system : syndromes and miscellaneous diseases; Risk factors; Sequence Analysis, DNA; Society; T cells; T-Lymphocyte Subsets - metabolism; Mexico; Idiopathic Pulmonary Fibrosis; Major Histocompatibility Complex; Hypersensitivity Pneumonitis; Idiopathic Pulmonary Fibrosis Patient; Alveolar Epithelial Cell; Lung disease; Pulmonary fibrosis; Respiratory disease; Idiopathic; MICA ligand; Genetics; Polymorphism; Biological receptor