The neuropeptide NMU amplifies ILC2-driven allergic lung inflammation - Wallrapp, Antonia; Riesenfeld, Samantha J.; Burkett, Patrick R.; Abdulnour, Raja-Elie E.; Nyman, Jackson; Dionne, Danielle; Hofree, Matan; Cuoco, Michael S.; Rodman, Christopher; Farouq, Daneyal; Haas, Brian J.; Tickle, Timothy L.; et al. (Nature (London), 2017)
Keywords: 14/63; 38/91; 631/250/2504/2506; 631/337/2019; 64/60; Allergens; Allergies; Allergy; Animals; Asthma; Cytokines; Female; Gene expression; Gene Expression Regulation; Gene sequencing; Generalized linear models; Genomics; Homeostasis; Humanities and Social Sciences; Hypersensitivity; Hypersensitivity - immunology; Hypersensitivity - pathology; Immunity, Innate - immunology; Inflammation; Inflammation - immunology; Inflammation - pathology; Interleukin-17 - immunology; Interleukin-33 - immunology; Ligands; Lung - immunology; Lung - pathology; Lung diseases; Lungs; Lymphocytes - immunology; Lymphoid cells; Male; Mice; Mice, Inbred C57BL; Mucosa; multidisciplinary; Neuromedin; Neuropeptides; Neuropeptides - metabolism; Physiological aspects; Receptors, Neurotransmitter - biosynthesis; Receptors, Neurotransmitter - genetics; Receptors, Neurotransmitter - metabolism; Respiratory Mucosa - immunology; Respiratory Mucosa - pathology; Ribonucleic acid; RNA; Science; Signal Transduction; Signaling; Stimulation; Subpopulations; Transcription activation; Transcription, Genetic; United States